MIR324: A Non-Coding RNA Molecule in Post-Transcriptional Gene Regulation and Cellular Signaling Pathways
MIR324: A Non-Coding RNA Molecule in Post-Transcriptional Gene Regulation and Cellular Signaling Pathways
MicroRNA (miRNA) 324 (MIR324) is a non-coding RNA molecule that plays a critical role in various biological processes. MIR324 is a member of the 21 non-coding RNAs (ncRNAs) family, which are known as miRNAs. These small non -coding RNAs are derived from functional RNA viruses and have been shown to play important roles in post-transcriptional gene regulation, gene expression, and cell survival.
MIR324 is a 24.1 kilobase (kb) RNA molecule and has a unique 5' end that contains a 7-base exon and a 3' end that contains a stem-loop structure. The 3' end of MIR324 contains a unique double-stranded structure that consists of an alternating between GAG and AUG codons, and this region is known as the loop A.
MIR324 has been shown to have a wide range of functions in various biological processes, including cell growth, apoptosis, and metabolism. One of the well-documented functions of MIR324 is its role in cell apoptosis. MIR324 has been shown to be a negative regulator of cell apoptosis, which means that when MIR324 is expressed in cells, it can prevent apoptosis from occurring.
In addition to its role in cell apoptosis, MIR324 has also been shown to play a critical role in cell metabolism. For example, MIR324 has been shown to be involved in the regulation of cellular energy metabolism, and it has been shown to interact with several different cellular signaling pathways, including the TOR signaling pathway.
MIR324 has also been shown to play a role in the regulation of gene expression. For example, MIR324 has been shown to interact with the transcription factor E2F1, and it has been shown to play a role in the regulation of E2F1-mediated gene expression.
MIR324 has also been shown to have a role in the regulation of cellular interactions with the extracellular matrix (ECM). For example, MIR324 has been shown to be involved in the regulation of cell-ECM interactions, and it has been shown to interact with the ECM protein, N-cadherin.
MIR324 has also been shown to play a role in the regulation of cellular signaling pathways that are involved in cell adhesion, migration and invasion. For example, MIR324 has been shown to be involved in the regulation of the cadherin-based signaling pathway, and it has been shown to interact with the cadherin protein.
MIR324 has also been shown to play a role in the regulation of the cell cycle. For example, MIR324 has been shown to be involved in the regulation of the G1/S transition, and it has been shown to interact with the protein, p21.
MIR324 has also been shown to play a role in the regulation of cellular stress response. For example, MIR324 has been shown to be involved in the regulation of the cellular stress response, and it has been shown to interact with the protein, p16INK4a.
MIR324 has also been shown to play a role in the regulation of the development and progression of cancer. For example, MIR324 has been shown to be involved in the regulation of cancer cell survival, and it has been shown to interact with the protein, p53.
MIR324 has also been shown to play a role in the regulation of the immune response. For example, MIR324 has been shown to be involved in the regulation of the immune response, and it has been shown to interact with the protein,
Protein Name: MicroRNA 324
The "MIR324 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR324 comprehensively, including but not limited to:
• general information;
• protein structure and compound binding;
• protein biological mechanisms;
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• expression level;
• disease relevance;
• drug resistance;
• related combination drugs;
• pharmacochemistry experiments;
• related patent analysis;
• advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai
More Common Targets
MIR325 | MIR325HG | MIR326 | MIR328 | MIR329-1 | MIR329-2 | MIR330 | MIR331 | MIR335 | MIR337 | MIR338 | MIR339 | MIR33A | MIR33B | MIR340 | MIR342 | MIR345 | MIR346 | MIR34A | MIR34AHG | MIR34B | MIR34C | MIR3529 | MIR3591 | MIR3605 | MIR3606 | MIR3609 | MIR361 | MIR3610 | MIR3611 | MIR3612 | MIR3613 | MIR3614 | MIR3615 | MIR3616 | MIR3617 | MIR3618 | MIR3619 | MIR362 | MIR3620 | MIR3621 | MIR3622A | MIR3622B | MIR363 | MIR3646 | MIR3648-1 | MIR3649 | MIR3650 | MIR3651 | MIR3652 | MIR3654 | MIR3655 | MIR3658 | MIR3659 | MIR365A | MIR365B | MIR3660 | MIR3661 | MIR3662 | MIR3663 | MIR3664 | MIR3665 | MIR3667 | MIR3667HG | MIR3668 | MIR367 | MIR3670-1 | MIR3670-2 | MIR3671 | MIR3672 | MIR3674 | MIR3675 | MIR3677 | MIR3678 | MIR3679 | MIR3680-1 | MIR3680-2 | MIR3681 | MIR3681HG | MIR3682 | MIR3683 | MIR3685 | MIR3686 | MIR3687 | MIR3688-1 | MIR3688-2 | MIR3689A | MIR3689B | MIR3689C | MIR3689D1 | MIR3689D2 | MIR3689F | MIR369 | MIR3690 | MIR3691 | MIR3692 | MIR370 | MIR3713 | MIR3714 | MIR371A
