Target Name: MIR524
NCBI ID: G574478
Review Report on MIR524 Target / Biomarker Content of Review Report on MIR524 Target / Biomarker
MIR524
Other Name(s): microRNA 524 | MIRN524 | hsa-miR-524-5p | hsa-mir-524 | hsa-miR-524-3p | mir-524 | MicroRNA 524

miRNA 524: A Non-Coding RNA Molecule and Its Potential Therapeutic Applications

MicroRNA (miRNA) 524 is a non-coding RNA molecule that plays a crucial role in various cellular processes. It is a part of the miRNA family, which consists of approximately 200 non-coding RNAs that have been shown to regulate gene expression in various organisms, including humans. One of the unique features of miRNA 524 is its expression pattern, which is highly dependent on the cell type and the context in which it is expressed.

MiRNA 524 has been shown to play a key role in the regulation of cell proliferation, apoptosis, and various signaling pathways. It is expressed in various tissues and cells throughout the body, including the brain, pancreas, and immune cells. It is also known to be involved in the regulation of cellular processes that are critical for human health, such as the development and progression of cancer, and the regulation of insulin sensitivity.

One of the reasons why miRNA 524 is of interest as a drug target is its involvement in the regulation of cell proliferation. miRNA 524 has been shown to play a negative role in the regulation of cell proliferation by inhibiting the activities of the cell cycle kinases, which are responsible for the cyclical division of cells. This means that when miRNA 524 is expressed at high levels, it can inhibit the ability of cells to divide and proliferate, which can lead to a reduction in the number of cancer cells that can be produced.

Another potential drug target for miRNA 524 is its role in the regulation of apoptosis, which is the process by which cells decide when to die and are programmed to undergo cell death. MiRNA 524 has been shown to play a positive role in the regulation of apoptosis by promoting the activities of the apoptosis-associated protein (AP-120), which is involved in the formation of executable intracellular granules (EXIGs) that contain apoptotic enzymes. This means that when miRNA 524 is expressed at high levels, it can increase the production of EXIGs and promote the formation of apoptotic cells, which can be a potential therapeutic target for cancer treatment.

MiRNA 524 is also of interest as a potential biomarker for various diseases, including cancer, neurodegenerative diseases, and autoimmune diseases. Its expression patterns can be affected by various factors, including genetic and environmental factors, which can affect its levels and its expression. This makes it a potential source of biomarkers that can be used to diagnose and monitor these diseases.

In conclusion, miRNA 524 is a non-coding RNA molecule that plays a crucial role in various cellular processes. Its expression pattern is highly dependent on the cell type and context, and it has been shown to play a key role in the regulation of cell proliferation, apoptosis, and various signaling pathways. As a result, miRNA 524 is of interest as a potential drug target and biomarker for various diseases. Further research is needed to fully understand its mechanisms of action and its potential therapeutic applications.

Protein Name: MicroRNA 524

The "MIR524 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR524 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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