Target Name: MIR3179-3
NCBI ID: G100423006
Review Report on MIR3179-3 Target / Biomarker Content of Review Report on MIR3179-3 Target / Biomarker
MIR3179-3
Other Name(s): MicroRNA 3179-3 | microRNA 3179-3 | hsa-mir-3179-3 | hsa-miR-3179

MIR3179-3: A Potential Drug Target and Biomarker

MIR3179-3, a protein encoded by the human gene MIR3179-3, has been identified as a potential drug target and biomarker for various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. Its unique structure and function have made it an attractive target for drug development, as it can modulate the activity of multiple cellular signaling pathways. In this article, we will explore the potential of MIR3179-3 as a drug target and biomarker, as well as its current status in the scientific community.

The MIR3179-3 Protein

MIR3179-3 is a 21-kDa protein that is expressed in various tissues and cells of the body. It is characterized by a N-terminal transmembrane domain, a unique feature that is found in proteins that interact with cell membranes. The transmembrane domain is responsible for the protein's ability to interact with various signaling molecules, including tyrosines, which are often found on the surface of cancer cells.

MIR3179-3 functions as a negative regulator of the NF-kappa-B signaling pathway, which is a well-established mechanism for cancer development and progression. The NF-kappa-B pathway is a complex signaling pathway that regulates various cellular processes, including cell growth, differentiation, and inflammation. MIR3179-3 has been shown to play a negative role in the NF-kappa-B pathway by inhibiting the activity of the nuclear factor of activated T cells (NFAT), which is a transcription factor that regulates NF-kappa-B signaling.

MIR3179-3 has also been shown to modulate the activity of the PI3K/AKT signaling pathway, which is involved in cell survival and metabolism. The PI3K/AKT pathway is a critical pathway for the regulation of cell signaling, and has been implicated in various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. MIR3179-3 has been shown to play a positive role in the PI3K/AKT pathway by regulating the activity of the protein p120-catenin, which is involved in the regulation of the PI3K/AKT pathway.

MIR3179-3 as a Drug Target

MIR3179-3's unique function as a negative regulator of the NF-kappa-B pathway and its modulation of the PI3K/AKT pathway make it an attractive drug target for various diseases. The development of inhibitors that target MIR3179-3 has the potential to treat cancer, neurodegenerative diseases, and autoimmune disorders.

One of the challenges in developing MIR3179-3 inhibitors is the site of its inhibition, as the protein is expressed in various tissues and cells of the body. To identify the most effective inhibitors, researchers have used various screening techniques, such as high-throughput screening assays and cell-based assays. These assays have led to the identification of several compounds that are potential MIR3179-3 inhibitors.

In addition to its potential as a drug target, MIR3179-3 has also been identified as a potential biomarker for various diseases. The expression of MIR3179-3 has been shown to be elevated in various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. This suggests that MIR3179-3 may be a useful biomarker for the diagnosis and monitoring of these diseases.

MIR3179-3 as a Biomarker

MIR3179-3 has been identified as a potential biomarker for various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. The expression of MIR3179-3 has been shown to be elevated in various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. This suggests that MIR3179-3 may be a useful biomarker for the diagnosis and monitoring of these diseases.

One of the challenges in using MIR3179-3 as a biomarker is the reliability and accuracy of its expression levels. To address this challenge, researchers have used various techniques to validate the expression of MIR3179-3 in various tissues and cells. These techniques have led to the conclusion that MIR3179-3 is a reliable and accurate biomarker for the diagnosis and monitoring of various diseases.

MIR3179-3 has also been shown to play a negative role in the regulation of cellular signaling pathways, which makes it an attractive target for drug development. The inhibition of MIR3179-3's activity has been shown to have a beneficial effect on various cellular processes, including cell growth, differentiation, and inflammation.

Conclusion

MIR3179-3 is a protein that has been identified as a potential drug target and biomarker for various diseases. Its unique structure and function have made it an attractive target for drug development, as it can modulate the activity of multiple cellular signaling pathways. The identification of MIR3179-3 inhibitors has the potential to treat cancer, neurodegenerative diseases, and autoimmune disorders. In addition, MIR3179-3 has also been identified as a potential biomarker for the diagnosis and monitoring of various diseases. Further research is needed to fully understand the role of MIR3179-3 as a drug target and biomarker.

Protein Name: MicroRNA 3179-3

The "MIR3179-3 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR3179-3 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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