Target Name: MIR566
NCBI ID: G693151
Review Report on MIR566 Target / Biomarker Content of Review Report on MIR566 Target / Biomarker
MIR566
Other Name(s): MicroRNA 566 | MIRN566 | hsa-mir-566

MIR566: A Non-Coding RNA Molecule Regulating Gene Expression and Disease

MicroRNA 566 (miRNA 566) is a non-coding RNA molecule that plays a critical role in various biological processes in the cell. It is a key regulator of gene expression, and is involved in the development, maintenance, and regression of tissues and organs . MIR566 is coded in the gene HOMER-C1723 and has 19 amino acid residues.

MIR566 is a member of the miRNA family, which are small non-coding RNAs that play a central role in post-transcriptional gene regulation. These RNAs act as scaffolds to recruit chromatin-remodeling complexes, and are involved in the regulation of chromatin accessibility , gene expression, and cellular processes such as cell growth, apoptosis, and autophagy.

MIR566 is expressed in a variety of tissues and organs, including brain, heart, liver, and muscle. It is highly expressed in the brain, where it is the most highly expressed gene in the brain. It is also expressed in other organs, including the heart, liver, and muscle.

MIR566 is involved in the regulation of gene expression and has been implicated in the development and progression of various diseases. For example, it has been shown to be involved in the development of cancer, and to play a role in the regulation of cell apoptosis.

In addition to its involvement in disease, MIR566 has also been shown to have potential as a drug target. Several studies have identified potential drug targets for MIR566, including those involved in cell signaling pathways, cell cycle regulation, and apoptosis.

One potential drug target for MIR566 is the PI3K/AKT signaling pathway. This pathway is involved in the regulation of cell proliferation, survival, and angiogenesis, and is a target for several anti-cancer drugs. MIR566 has been shown to be involved in the regulation of PI3K/AKT signaling, and has been shown to inhibit the activity of the PI3K/AKT signaling pathway.

Another potential drug target for MIR566 is the p53 signaling pathway. This pathway is involved in the regulation of DNA damage repair, and is a target for several anti-cancer drugs. MIR566 has been shown to be involved in the regulation of p53 signaling, and has been shown to enhance the activity of the p53 signaling pathway.

In addition to its potential as a drug target, MIR566 has also been shown to have potential as a biomarker. The expression of MIR566 has been shown to be associated with the development of certain diseases, such as cancer and neurodegenerative diseases. Additionally, MIR566 has has been shown to be involved in the regulation of cellular processes, such as cell growth, apoptosis, and autophagy, which could be potential biomarkers for certain diseases.

Overall, MIR566 is a promising molecule for drug development as it has been shown to be involved in several cellular processes that are involved in the development and progression of diseases. Further research is needed to fully understand the role of MIR566 in disease and to identify potential drug targets and biomarkers.

Protein Name: MicroRNA 566

The "MIR566 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR566 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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