Target Name: MIR587
NCBI ID: G693172
Review Report on MIR587 Target / Biomarker Content of Review Report on MIR587 Target / Biomarker
MIR587
Other Name(s): MicroRNA 587 | hsa-mir-587 | microRNA 587 | MIRN587 | hsa-miR-587

MIR587: A Non-Coding RNA Molecule as A Potential Drug Target and Biomarker

MicroRNA (miRNA) 587 is a non-coding RNA molecule that has been identified as a potential drug target and biomarker for various diseases, including cancer. MIR587 is a small molecule that can interact with specific miRNA targets, known as target miRNAs, to either enhance or decrease their levels in the cell. This interaction between MIR587 and target miRNAs can lead to the regulation of gene expression, which can have a significant impact on cellular processes such as cell growth, apoptosis, and inflammation.

One of the unique features of MIR587 is its ability to interact with multiple miRNA targets. This interaction allows MIR587 to be used as a drug or biomarker in a variety of settings, including cancer treatment and diagnosis. For example, MIR587 has been shown to reduce the levels of miR18a, a well-known cancer-promoting miRNA, in cancer cells. This reduction in miR18a levels has been associated with increased cell cycle arrest and a reduction in cell proliferation, making MIR587 an attractive potential drug target for cancer treatment.

Another potential application of MIR587 is its ability to enhance the levels of miR123, a miRNA that has been shown to promote cell apoptosis in cancer cells. By increasing the levels of miR123, MIR587 has been shown to promote cell death, which can be an effective way to target cancer cells. Additionally, MIR587 has been shown to increase the levels of miR16, a miRNA that has been associated with cancer stem cell maintenance, which can further enhance the potential for cancer treatment.

MIR587 has also been shown to interact with multiplemiR1, a miRNA that has been associated with cell cycle progression and the development of cancer. The interaction between MIR587 and miR1 has been shown to result in increased levels of miR1 in the cell, which can further enhance the potential for cancer treatment.

In addition to its potential as a drug target, MIR587 has also been identified as a potential biomarker for cancer. The levels of MIR587 have been shown to be reduced in a variety of cancer types, including breast, lung, and ovarian cancer. This reduction in MIR587 levels has been associated with poor prognosis and increased cancer recurrence. Additionally, MIR587 has been shown to be downregulated in cancer cells, which can make it an attractive biomarker for cancer diagnosis and monitoring.

MIR587 is a small molecule that can interact with multiple miRNA targets, leading to the regulation of gene expression and the regulation of cellular processes such as cell growth, apoptosis, and inflammation. Its ability to interact with multiple miRNA targets and its potential as a drug target and biomarker make MIR587 an attractive target for research and development in a variety of diseases, including cancer. Further studies are needed to fully understand the mechanism of MIR587 and its potential as a drug and biomarker.

Protein Name: MicroRNA 587

The "MIR587 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR587 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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