Target Name: MIR6761
NCBI ID: G102465456
Review Report on MIR6761 Target / Biomarker Content of Review Report on MIR6761 Target / Biomarker
MIR6761
Other Name(s): microRNA 6761 | MicroRNA 6761 | hsa-mir-6761 | hsa-miR-6761-5p | hsa-miR-6761-3p

miRNA 6761: A Potential Drug Target and Biomarker for Cell Apoptosis and Metabolism

MicroRNA (miRNA) 6761 is a non-coding RNA molecule that has been identified as a potential drug target and biomarker for various diseases, including cancer. Mammalian miRNAs are small non-coding RNAs that are derived from microRNAs, and they play a critical role in post-transcriptional gene regulation by binding to specific targetmRNAs.

MiRNA 6761 is a member of the miRNA 21 family, which is known for its involvement in various cellular processes, including cell growth, apoptosis, and metabolism. The miRNA 21 family has been implicated in the regulation of various cellular processes, including cell cycle progression, cell survival, and cell angiogenesis.

MiRNA 6761 has been shown to play a critical role in the regulation of cell apoptosis, which is the process by which cells die and are removed from the body. In fact, MIR6761 has been shown to be involved in the regulation of cell apoptosis in various cell types, including cancer cells.

One of the key mechanisms by which miRNA 6761 regulates cell apoptosis is through its ability to bind to specific targetmRNAs. This is accomplished through the use of a process called post-transcriptional regulation, which involves the addition of specific RNA molecules to the RNA polymerase II complex after the primary RNA has been synthesized.

MiRNA 6761 has been shown to interact with several different targetmRNAs, including the transcription factor E2F1, which is involved in the regulation of various cellular processes, including cell growth, apoptosis, and metabolism. Additionally, miRNA 6761 has been shown to interact with the translation factor IFNIR1, which is involved in the regulation of protein synthesis and cell growth.

In addition to its role in cell apoptosis, miRNA 6761 has also been shown to play a critical role in the regulation of cell metabolism and energy homeostasis. This is accomplished through its ability to interact with the energy-producing enzyme Pyruvate Carrier Proteins (PCP), which is involved in the transport of pyruvate across the cell membrane and its metabolism.

MiRNA 6761 has also been shown to play a critical role in the regulation of cell-cell interactions and tissue architecture. This is accomplished through its ability to interact with the cell-cell adhesion molecule E-cadherin, which is involved in the regulation of cell-cell adhesion and tissue architecture.

In conclusion, miRNA 6761 is a non-coding RNA molecule that has been shown to play a critical role in the regulation of cell apoptosis, cell metabolism, and cell-cell interactions. Its potential as a drug target or biomarker makes it an attractive target for further research into the mechanisms of post-transcriptional gene regulation and the regulation of various cellular processes. Further studies are needed to fully understand the role of miRNA 6761 in the regulation of cell processes and its potential as a drug or biomarker.

Protein Name: MicroRNA 6761

The "MIR6761 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR6761 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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