Target Name: MIR6828
NCBI ID: G102465497
Review Report on MIR6828 Target / Biomarker Content of Review Report on MIR6828 Target / Biomarker
MIR6828
Other Name(s): hsa-mir-6828 | hsa-miR-6828-3p | microRNA 6828 | MicroRNA 6828 | hsa-miR-6828-5p

MIR6828: A Potential Drug Target and Biomarker for treatable Brain Disorders

MIR6828 is a non-coding RNA molecule that has been identified as a potential drug target and biomarker for treatable brain disorders. The molecule is derived from the heat shock protein (HSP) gene and has been shown to play a critical role in the regulation of gene expression and protein synthesis in various organisms, including humans. MIR6828 has been shown to be involved in a wide range of physiological processes, including cell growth, apoptosis, and inflammation.

Disease-related changes in gene expression

MIR6828 has been shown to be involved in the regulation of gene expression in response to various diseases, including neurodegenerative disorders, cancer, and inflammation. For example, studies have shown that MIR6828 is downregulated in the brains of individuals with Alzheimer's disease, and that this downregulation is associated with increased neurofibrillary tangles and decreased protein synthesis.

MIR6828 as a potential drug target

MIR6828 has been identified as a potential drug target due to its involvement in the regulation of gene expression in response to various diseases. The molecule has been shown to play a critical role in the regulation of protein synthesis and has been shown to be involved in the development of neurodegenerative disorders.

MIR6828 has also been shown to be involved in the regulation of apoptosis, which is a natural process that helps to remove damaged or dysfunctional cells from the body. Studies have shown that MIR6828 is involved in the regulation of apoptosis in various organisms, including humans.

MIR6828 as a biomarker

MIR6828 has also been identified as a potential biomarker for a wide range of diseases, including neurodegenerative disorders, cancer, and cardiovascular disease. Studies have shown that MIR6828 is expressed in various tissues and cells, including brain, spinal cord, and peripheral tissues, and that it is involved in the regulation of gene expression in response to various diseases.

MIR6828 as a potential therapeutic approach

The identification of MIR6828 as a potential drug target and biomarker for various diseases has led to a new approach to the development of therapeutic compounds. Studies have shown that MIR6828 can be targeted with small molecules, peptides, and antibodies to selectively modulate its activity and improve its function as a drug target or biomarker.

Conclusion

MIR6828 is a non-coding RNA molecule that has been identified as a potential drug target and biomarker for treatable brain disorders. The molecule is derived from the heat shock protein (HSP) gene and has been shown to play a critical role in the regulation of gene expression and protein synthesis in various organisms, including humans. Further studies are needed to fully understand the role of MIR6828 in the regulation of gene expression and protein synthesis in various diseases, as well as its potential as a drug target and biomarker.

Protein Name: MicroRNA 6828

The "MIR6828 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR6828 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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