Target Name: MIR489
NCBI ID: G574442
Review Report on MIR489 Target / Biomarker Content of Review Report on MIR489 Target / Biomarker
MIR489
Other Name(s): MicroRNA 489 | microRNA 489 | hsa-mir-489 | MIRN489 | hsa-miR-489-5p | mir-489 | hsa-miR-489-3p

MIR489: A Non-Coding RNA Molecule with Potential Drug or Biomarker Targets

MicroRNA (miRNA) 489 (MIR489) is a non-coding RNA molecule that plays a critical role in various biological processes in humans. MIR489 is a member of the miRNA family, which consists of approximately 200 non-coding RNAs that are involved in regulating gene expression. MIR489 is expressed in various tissues and cells of the body and is involved in a wide range of physiological processes, including cell growth, differentiation, and apoptosis.

One of the unique features of MIR489 is its structure. MIR489 is a single-stranded RNA molecule that has a length of approximately 24 amino acid residues. It has a highly conserved conserved sequence, including a conserved tail domain at the 5' end and 3' end each have a conserved amino acid residue. The conserved nature of MIR489 makes it a potential drug or biomarker target.

MIR489 is a key regulator of cell growth and differentiation. It has been shown to be involved in the regulation of various cellular processes, including cell cycle progression, cell adhesion, and cell survival. MIR489 has been shown to play a role in the regulation of cell proliferation and has been shown to be involved in the development of various types of cancer.

MIR489 is also involved in the regulation of apoptosis. Apoptosis is a critical regulatory process that helps maintain tissue homeostasis and removes damaged or dysfunctional cells. MIR489 has been shown to be involved in the regulation of apoptosis and has been shown to play a role in the development of neurodegenerative diseases.

MIR489 has also been shown to be involved in the regulation of inflammation. Inflammation is a critical biological response to tissue damage and infection. MIR489 has also been shown to be involved in the regulation of inflammation and has been shown to play a role in the development of various inflammatory diseases.

MIR489 is also involved in the regulation of cellular signaling pathways. It has been shown to be involved in the regulation of various cellular signaling pathways, including the TGF-β signaling pathway. TGF-β is a critical signaling pathway that is involved in the regulation of cell growth, differentiation, and apoptosis.

MIR489 has also been shown to be involved in the regulation of gene expression. It has been shown to be a negative regulator of several genes and has been shown to play a role in the regulation of gene expression. This suggests that MIR489 may be a useful drug target or biomarker for the development of various diseases.

In conclusion, MIR489 is a non-coding RNA molecule that plays a critical role in various biological processes in humans. Its unique structure and conservation make it a potential drug target or biomarker. Further research is needed to fully understand the role of MIR489 in various biological processes and to develop effective therapies or biomarkers based on it.

Protein Name: MicroRNA 489

The "MIR489 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR489 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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