Target Name: MIR4700
NCBI ID: G100616329
Review Report on MIR4700 Target / Biomarker Content of Review Report on MIR4700 Target / Biomarker
MIR4700
Other Name(s): microRNA 4700 | MicroRNA 4700 | hsa-mir-4700 | mir-4700 | hsa-miR-4700-3p | hsa-miR-4700-5p

MIR4700: A Potential Drug Target and Biomarker

MIR4700 is a non-coding RNA molecule that has been identified as a potential drug target and biomarker for various diseases, including cancer. Its unique structure and function have made it an attractive target for researchers to investigate, and its potential as a drug may have significant implications for the treatment of certain diseases.

MIR4700 is a microRNA (miRNA) that is expressed in various tissues and cells throughout the body. It is characterized by its 20- amino acid length and its unique stem-loop structure. MIR4700 has been shown to play a role in various cellular processes, including cell growth, apoptosis, and inflammation.

One of the most significant functions of MIR4700 is its ability to regulate the expression of other genes. This is achieved through its interactions with various protein molecules, including transcription factors. MIR4700 has been shown to interact with several transcription factors, including NF-kappa-B, AP-1, and STAT3. These interactions allow MIR4700 to regulate the expression of genes that are critical for cellular processes such as cell growth, apoptosis, and inflammation.

MIR4700 has also been shown to play a role in the regulation of cellular apoptosis. Apoptosis is a natural process that is essential for the development and maintenance of tissues and organs. However, when cells are no longer needed or are damaged, apoptosis can become uncontrolled and can lead to the formation of cancerous tumors. MIR4700 has been shown to regulate the apoptosis-associated protein (AP-1) in cancer cells. This regulation allows MIR4700 to promote the survival of cancer cells and contribute to their resistance to chemotherapy.

In addition to its role in apoptosis, MIR4700 has also been shown to play a role in cell signaling. It has been shown to interact with several signaling molecules, including TGF-β1, Wnt, and Notch. These interactions allow MIR4700 to regulate cellular processes such as cell proliferation, differentiation, and survival.

MIR4700 has also been shown to be involved in the regulation of inflammation. Chronic inflammation can contribute to a variety of diseases, including cancer. MIR4700 has been shown to play a role in the regulation of pro-inflammatory pathways, including the production of pro-inflammatory cytokines. This regulation allows MIR4700 to contribute to the maintenance of chronic inflammation and the development of cancer.

In conclusion, MIR4700 is a promising drug target and biomarker for various diseases. Its unique structure and function, as well as its ability to regulate the expression of other genes and play a role in cellular processes such as apoptosis, signaling, and inflammation, make it an attractive target for researchers to investigate. Further studies are needed to fully understand the potential of MIR4700 as a drug and to determine its effectiveness in treating various diseases.

Protein Name: MicroRNA 4700

The "MIR4700 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR4700 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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