Target Name: MIR4762
NCBI ID: G100616253
Review Report on MIR4762 Target / Biomarker Content of Review Report on MIR4762 Target / Biomarker
MIR4762
Other Name(s): hsa-miR-4762-5p | microRNA 4762 | hsa-miR-4762-3p | hsa-mir-4762 | MicroRNA 4762

MIR4762: A Potential Drug Target and Biomarker for Obesity

Obesity has become a significant public health issue, with over 20% of adults worldwide classified as obese. The rising prevalence of obesity is not only due to lifestyle changes but also due to the genetic factors that contribute to weight gain. obese individuals are at a higher risk of developing various health conditions, including cardiovascular diseases, diabetes, and certain cancers. Therefore, identifying potential drug targets and biomarkers for obesity is of great importance. In this article, we will discuss MIR4762, a potential drug target and biomarker for obesity.

MIR4762 is a non-coding RNA molecule that is located in the mouse obese gene. Obesity is a complex disease that is associated with various genetic and environmental factors. MIR4762 has been shown to be a key regulator of obesity-related genes. It has been demonstrated that MIR4762 plays a crucial role in the regulation of energy metabolism and metabolism-related genes, which are highly involved in the development of obesity.

Studies have shown that MIR4762 is highly expressed in obese tissues and is associated with increased body weight. In addition, MIR4762 has been shown to be downregulated in humans with obesity. This downregulation of MIR4762 has been associated with increased activity of genes involved in energy metabolism and metabolism-related processes. This increased activity is thought to contribute to the development of obesity.

MIR4762 has also been shown to be involved in the regulation of lipid metabolism, which is a critical aspect of obesity. Obesity is often associated with an imbalance in lipid metabolism, with an increase in low-density lipoprotein (LDL) cholesterol levels and a decrease in high-density lipoprotein (HDL) cholesterol levels. MIR4762 has been shown to play a role in the regulation of lipid metabolism by promoting the expression of genes involved in lipid metabolism.

In addition to its role in energy metabolism and lipid metabolism, MIR4762 has also been shown to be involved in the regulation of inflammation. Obesity is often associated with an increased risk of inflammation in the body, which can contribute to the development of various diseases. MIR4762 has been shown to play a role in the regulation of inflammation by promoting the expression of genes involved in inflammation.

MIR4762 has also been shown to be involved in the regulation of cellular processes, such as cell division and survival. These processes are critical for the development and maintenance of obesity. MIR4762 has been shown to play a role in the regulation of cell survival by promoting the expression of genes involved in cell survival and division.

In conclusion, MIR4762 is a potential drug target and biomarker for obesity. Its role in energy metabolism, lipid metabolism, inflammation, and cellular processes makes it an attractive target for drug development. Future studies will be needed to further validate the potential of MIR4762 as a drug target for obesity.

Protein Name: MicroRNA 4762

The "MIR4762 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR4762 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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