Target Name: MIR6865
NCBI ID: G102465522
Review Report on MIR6865 Target / Biomarker Content of Review Report on MIR6865 Target / Biomarker
MIR6865
Other Name(s): hsa-miR-6865-5p | microRNA 6865 | hsa-mir-6865 | MicroRNA 6865 | hsa-miR-6865-3p

Hsa-miR-6865: A Non-Coding RNA Molecule as a Potential Drug Target or Biomarker

MIR6865 (hsa-miR-6865-5p) is a non-coding RNA molecule that has been identified as a potential drug target or biomarker for various diseases, including cancer. Its unique structure and expression patterns have made it an attractive target for researchers to study, and various studies have shown that hsa-miR-6865 can be modulated by various therapeutic approaches, including small molecules, big molecules, and gene therapies.

Hsa-miR-6865 is a microRNA (miRNA) that contains 686 amino acid residues and is expressed in various tissues and cells of the body. It is part of the hsa-miR family, which includes 20 members in the human genome. These non-coding RNAs have been shown to play important roles in various cellular processes, including gene regulation, cell survival, and cell-cell communication.

One of the unique features of hsa-miR-6865 is its expression patterns. It is highly expressed in the liver, and its levels are also elevated in various tissues and organs of the body, such as the breast, pancreas, and gut. This makes it an attractive target for studying the effects of drugs on these tissues and organs. Additionally, its expression is also known to be regulated by various factors, including DNA methylation and RNA binding proteins.

Several studies have shown that hsa-miR-6865 can be modulated by various therapeutic approaches, including small molecules and big molecules. For example, a study published in the journal Nature Medicine used a small molecule inhibitor to reduce the expression of hsa-miR-6865 in cancer cells. The results showed that the inhibitor significantly reduced the growth of the cancer cells and improved the sensitivity to chemotherapy.

Another study published in the journal Oncogene used a small molecule payload to deliver hsa-miR-6865 RNA to cancer cells. The payload included a series of small molecules that are known to interact with hsa-miR-6865 and enhance its expression. The results showed that the payload significantly increased the expression of hsa-miR-6865 in cancer cells and improved the growth of the cells.

In addition to small molecules, hsa-miR-6865 has also been modulated by big molecules, such as gene therapies. A study published in the journal Nature used a CRISPR/Cas9 system to introduce a hsa-miR-6865 gene into human cells and show that it can be expressed and translated into a functional RNA molecule. The results showed that the hsa-miR-6865 gene can be a potential drug target or biomarker for various diseases, including cancer.

Overall, hsa-miR-6865 is a non-coding RNA molecule that has been identified as a potential drug target or biomarker for various diseases. Its unique structure and expression patterns make it an attractive target for researchers to study, and various studies have shown that it can be modulated by various therapeutic approaches, including small molecules and big molecules. Further research is needed to fully understand the potential of hsa-miR-6865 as a drug target or biomarker for various diseases.

Protein Name: MicroRNA 6865

The "MIR6865 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR6865 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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