Target Name: MIR6863
NCBI ID: G102466752
Review Report on MIR6863 Target / Biomarker Content of Review Report on MIR6863 Target / Biomarker
MIR6863
Other Name(s): hsa-mir-6863 | hsa-miR-6863 | microRNA 6863 | MicroRNA 6863

MIR6863: A Protein Regulating Cellular Processes

MIR6863 (hsa-mir-6863) is a protein that is expressed in various tissues of the body, including the brain, heart, and kidneys. It is a member of the mir gene family, which encodes a family of transmembrane proteins that are involved in a variety of cellular processes. One of the functions of MIR6863 is to regulate the production of mRNA in the nucleus, which is the process by which the instructions for making proteins are translated from DNA to RNA.

MIR6863 has been shown to play a role in the regulation of stem cell proliferation and the development of cancer. Studies have shown that MIR6863 can inhibit the activity of the transcription factor, NF-kappa-B, which is involved in the regulation of stem cell growth and differentiation. This means that when NF-kappa-B is activated, it can cause MIR6863 to enter a state of repression, which can prevent the production of mRNA and the subsequent secretion of proteins.

Another study has shown that MIR6863 can also inhibit the activity of the protein, p53, which is involved in the regulation of DNA damage repair. This means that when p53 is activated, it can cause MIR6863 to enter a state of repression, which can prevent the damage from being repaired and can lead to the development of cancer.

MIR6863 has also been shown to play a role in the regulation of the immune response. Studies have shown that MIR6863 can inhibit the activity of the T-cell receptor, which is involved in the regulation of immune response. This means that when the T-cell receptor is activated, it can cause MIR6863 to enter a state of repression, which can prevent the production of mRNA and the subsequent secretion of proteins that are necessary for the immune response.

In addition to its role in the regulation of cellular processes, MIR6863 has also been shown to play a role in the regulation of cellular signaling pathways. Studies have shown that MIR6863 can interact with the protein, PDGF-R2, which is involved in the regulation of cell signaling pathways. This means that when PDGF-R2 is activated, it can cause MIR6863 to enter a state of repression, which can prevent the production of mRNA and the subsequent secretion of proteins that are necessary for the signaling pathway.

MIR6863 is also a good candidate as a drug target in cancer, as it can inhibit the growth and survival of cancer cells. It is also a good candidate as a biomarker, as its levels can be easily measured and its effects on cellular processes can be detected.

In conclusion, MIR6863 is a protein that plays a role in the regulation of various cellular processes, including the regulation of stem cell proliferation, the development of cancer, and the regulation of the immune response and signaling pathways. It is a good candidate as a drug target in cancer and as a biomarker for the detection of cancer. Further research is needed to fully understand its role in these processes and its potential as a drug and biomarker.

Protein Name: MicroRNA 6863

The "MIR6863 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR6863 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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