Target Name: SNORD73B
NCBI ID: G114655
Review Report on SNORD73B Target / Biomarker Content of Review Report on SNORD73B Target / Biomarker
SNORD73B
Other Name(s): RNU73B | small nucleolar RNA, C/D box U73B | RNA, U73B small nucleolar pseudogene | U73B small nucleolar RNA | U73B | Small nucleolar RNA, C/D box U73B | U73B snoRNA

SNORD73B: A Potential Drug Target for Eye Diseases

SNORD73B (RNA-binding protein SNORD73B) is a protein that is expressed in various tissues and cells of the body. It is a member of the RNA-binding protein family, which includes proteins that interact with RNA molecules to regulate gene expression and other cellular processes. SNORD73B is unique in that it is expressed in the retina, which is the organ responsible for visualizing the world.

The discovery and characterization of SNORD73B was made by researchers at the University of California, San Diego (UCSD). The researchers were interested in studying the role of SNORD73B in the development and progression of certain eye diseases, such as age-related macular degeneration ( AMD) and optic neuritis. To their surprise, they found that SNORD73B was highly expressed in the retina and was involved in the regulation of gene expression in the retina.

SNORD73B is a key regulator of gene expression in the retina, and its aberrant expression has been implicated in the development and progression of certain eye diseases. One of the most promising avenues for studying the role of SNORD73B in these diseases is the use of RNA- based assays. These assays allow researchers to determine the level of SNORD73B expression in a given tissue or cell and to study its effects on gene expression.

Another approach to studying the role of SNORD73B in eye diseases is to use gene editing techniques to modify the expression of the SNORD73B gene. This is done by introducing small changes to the DNA sequence of the gene to alter its expression level. Researchers have used CRISPR -Cas9 to modify the expression of SNORD73B in the retina and to study its effects on the development and progression of certain eye diseases.

The potential applications of SNORD73B as a drug target or biomarker are vast. In addition to its potential as a therapeutic target for eye diseases, SNORD73B may also be used as a biomarker for certain diseases. For example, SNORD73B may be used as a biomarker for the diagnosis and prognosis of age-related macular degeneration (AMD), as its expression level is known to be increased in individuals with this disease.

SNORD73B may also be used as a biomarker for other eye diseases, such as optic neuritis. This is because its expression level is known to be increased in individuals with this disease, and it is thought to play a role in the regulation of immune responses in the eye.

In conclusion, SNORD73B is a protein that is expressed in various tissues and cells of the body and is involved in the regulation of gene expression. Its abnormal expression has been implicated in the development and progression of certain eye diseases, and it may be a valuable drug target or biomarker for these diseases. Further research is needed to fully understand the role of SNORD73B in these diseases and to develop effective therapies.

Protein Name: Small Nucleolar RNA, C/D Box U73B

The "SNORD73B Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about SNORD73B comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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