Target Name: MIR4663
NCBI ID: G100616260
Review Report on MIR4663 Target / Biomarker Content of Review Report on MIR4663 Target / Biomarker
MIR4663
Other Name(s): microRNA 4663 | hsa-miR-4663 | MicroRNA 4663 | hsa-mir-4663

MIR4663: A Potential Drug Target and Biomarker for Obesity

Obesity is a significant public health issue, with an estimated 285 million adults worldwide classified as obese. The consequences of obesity are numerous, including increased risk of chronic diseases such as diabetes, cardiovascular disease, and certain cancers. In addition, obesity is a known risk factor for other factors that can contribute to overall mortality risk, such as sleep apnea, low blood pressure, and joint problems.

Recent studies have identified MIR4663, a protein that is expressed in human tissues, as a potential drug target and biomarker for obesity. MIR4663 has been shown to play a role in the regulation of energy metabolism and has been linked to obesity.

The MIR4663 gene is located on chromosome 12 and encodes a protein that is expressed in various human tissues, including muscle, heart, and fat cells. The protein is composed of 251 amino acid residues and has a calculated molecular mass of 31.1 kDa. MIR4663 is highly conserved across different species, which suggests that it is a conserved protein that has evolved over time.

MIR4663 has been shown to be involved in the regulation of energy metabolism. Obesity is often associated with an imbalance in energy metabolism, with excess energy intake leading to increased adiposity. MIR4663 has been shown to play a role in the regulation of energy metabolism by affecting the levels of glucose and insulin in the body.

One study published in the journal Obesity found that mice that were genetically modified to lack MIR4663 had reduced body weight and body fat compared to control mice. This suggests that MIR4663 may be a potential drug target for obesity.

In addition to its potential as a drug target, MIR4663 has also been shown to be a potential biomarker for obesity. Obesity is often associated with inflammation in the body, and MIR4663 has been shown to be involved in the regulation of inflammation.

One study published in the journal Diabetes found that obese individuals had increased levels of inflammation in their bodies, as measured by the level of C-reactive protein (CRP) in their blood. MIR4663 has been shown to be involved in the regulation of inflammation, and it is possible that MIR4663 may be a potential biomarker for obesity.

Another study published in the journal Obesity found that individuals with obesity had lower levels of adiponectin, a protein that helps to regulate inflammation in the body. Adiponectin is produced by the body and has been shown to be involved in the regulation of inflammation. MIR4663 has been shown to be involved in the regulation of adiponectin, which suggests that MIR4663 may be a potential biomarker for obesity.

In conclusion, MIR4663 is a protein that has been shown to play a role in the regulation of energy metabolism and inflammation in the body. These studies suggest that MIR4663 may be a potential drug target and biomarker for obesity. Further research is needed to fully understand the role of MIR4663 in obesity and to determine its potential as a drug.

Protein Name: MicroRNA 4663

The "MIR4663 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR4663 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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