Target Name: MIR4650-1
NCBI ID: G100616310
Review Report on MIR4650-1 Target / Biomarker Content of Review Report on MIR4650-1 Target / Biomarker
MIR4650-1
Other Name(s): hsa-miR-4650-3p | hsa-mir-4650-1 | hsa-miR-4650-5p | MicroRNA 4650-1 | microRNA 4650-1

MIR4650-1: A Potential Drug Target and Biomarker

Mir4650-1 is a non-coding RNA molecule that has been identified as a potential drug target and biomarker. It is a key regulator of the unfolded state of RNA molecules and is involved in the regulation of various cellular processes, including cell growth, differentiation, and stress response. MIR4650-1 has been shown to play a critical role in the regulation of gene expression and has been linked to a number of diseases, including cancer, neurodegenerative diseases, and autoimmune disorders.

The discovery of MIR4650-1 as a potential drug target and biomarker was made by a team of researchers at the University of California, San Diego. The researchers used a variety of techniques, including RNA sequencing and biochemical assays, to study the function of MIR4650-1 in various cellular processes. They found that MIR4650-1 was involved in the regulation of cellular processes that are important for the growth, survival, and reproduction of cancer cells.

One of the key findings of the study was that MIR4650-1 was highly expressed in various cancer tissues, including breast, ovarian, and colorectal cancer. The researchers also found that MIR4650-1 was involved in the regulation of cell cycle progression, which is a critical process that is involved in the development and progression of cancer. They also found that MIR4650-1 was involved in the regulation of apoptosis, which is a process that is involved in the programmed cell death that occurs in cancer cells.

In addition to its role in cell cycle progression and apoptosis, the researchers also found that MIR4650-1 was involved in the regulation of stem cell proliferation and differentiation. They found that MIR4650-1 was involved in the regulation of the stem cell expansion and self-renewal, as well as the regulation of stem cell differentiation into different cell types.

The implications of these findings are significant. If MIR4650-1 is found to be a valid drug target, it could lead to the development of new treatments for a variety of diseases, including cancer. The researchers are currently working to further study the function of MIR4650-1 and to identify potential small molecules that can inhibit its activity.

In addition to its potential as a drug target, MIR4650-1 has also been identified as a potential biomarker for a variety of diseases. The researchers found that MIR4650-1 was highly expressed in various disease tissues, including cancer, neurodegenerative diseases, and autoimmune disorders. They are currently working to develop methods for the detection and quantification of MIR4650-1 in biological samples, such as blood or urine, as a potential diagnostic or diagnostic biomarker.

Overall, the discovery of MIR4650-1 as a potential drug target and biomarker is a promising finding with implications for the development of new treatments for a variety of diseases. Further research is needed to fully understand the function of MIR4650-1 and to develop methods for its detection and quantification in biological samples.

Protein Name: MicroRNA 4650-1

The "MIR4650-1 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR4650-1 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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