Target Name: MIR1184-1
NCBI ID: G100302111
Review Report on MIR1184-1 Target / Biomarker Content of Review Report on MIR1184-1 Target / Biomarker
MIR1184-1
Other Name(s): hsa-mir-1184-1 | hsa-miR-1184 | MIR1184 | MIRN1184 | microRNA 1184-1 | hsa-mir-1184 | MicroRNA 1184-1

MIR1184-1: A Drug Target / Disease Biomarker

MIR1184-1 is a protein that is expressed in various tissues of the body, including the brain, heart, lungs, and gastrointestinal tract. It is a member of the MIR family of transmembrane proteins, which are characterized by their ability to form channels through the cell membrane. MIR1184-1 has been shown to play a role in the regulation of pain perception and neuroinflammation.

One of the key functions of MIR1184-1 is its role in pain perception. MIR1184-1 has been shown to interact with a variety of neurotransmitters, including opioids and opioid receptors. This interaction suggests that MIR1184-1 may play a role in the regulation of pain perception.

In addition to its role in pain perception, MIR1184-1 has also been shown to be involved in the regulation of neuroinflammation. Neuroinflammation is a type of inflammation that occurs in the brain and other nervous system tissues, and it can be caused by a variety of factors, including injury, infection, and cancer. MIR1184-1 has been shown to play a role in the regulation of neuroinflammation by mechanisms such as the production of pro-inflammatory cytokines and the recruitment of immune cells to the site of inflammation.

MIR1184-1 has also been shown to interact with a variety of signaling pathways, including the TGF-β pathway. The TGF-β pathway is a well-established pathway that is involved in the regulation of cell growth, differentiation, and inflammation. MIR1184-1 has been shown to play a role in the regulation of TGF-β signaling by mechanisms such as the inhibition of TGF-β activity by the protein negatively regulated by MIR1184-1.

MIR1184-1 has also been shown to play a role in the regulation of cellular processes such as cell adhesion and migration. MIR1184-1 has been shown to interact with a variety of cell adhesion molecules, including cadherins and E-cadherins. This interaction suggests that MIR1184-1 may play a role in the regulation of cell adhesion and migration.

In addition to its role in pain perception, neuroinflammation, and cellular processes, MIR1184-1 has also been shown to have potential as a drug target. For example, MIR1184-1 has been shown to be a potential drug target for the treatment of pain disorders, including chronic pain and neuroinflammation. In addition, MIR1184-1 has been shown to be involved in the regulation of neuroinflammation, which suggests that it may be an effective target for the treatment of neuroinflammation-related diseases.

In conclusion, MIR1184-1 is a protein that has been shown to play a role in a variety of cellular processes, including pain perception, neuroinflammation, and cellular adhesion and migration. Its potential as a drug target makes it an attractive target for the development of new treatments for a variety of diseases. Further research is needed to fully understand the role of MIR1184-1 in the regulation of pain perception and neuroinflammation, and to develop effective treatments based on this protein.

Protein Name: MicroRNA 1184-1

The "MIR1184-1 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR1184-1 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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