Target Name: MIR1287
NCBI ID: G100302133
Review Report on MIR1287 Target / Biomarker Content of Review Report on MIR1287 Target / Biomarker
MIR1287
Other Name(s): MIRN1287 | mir-1287 | hsa-miR-1287-5p | hsa-mir-1287 | Hsa-mir-1287 | microRNA 1287 | MicroRNA 1287 | hsa-miR-1287-3p

MIR1287: A Potential Drug Target and Biomarker

MIR1287, also known as interleukin-128 (IL-128), is a cytokine that plays a crucial role in the regulation of immune responses and inflammation. It is expressed in various tissues and cells throughout the body, including immune cells, epithelial cells, and cancer cells. MIR1287 has been identified as a potential drug target and biomarker for various diseases, including cancer, autoimmune diseases, and chronic obstructive pulmonary disease (COPD).

MIR1287 functions as a potent regulator of T cell responses, promoting the development and activation of CD4+ T cells, while inhibiting the activity of CD8+ T cells. This cytokine is known to promote the production of anti-inflammatory cytokines, such as IL-10, and to regulate the production of pro-inflammatory cytokines, such as TNF-伪 and IL-12. MIR1287 has been shown to have a therapeutic effect on various diseases, including cancer, autoimmune diseases, and COPD.

One of the potential mechanisms by which MIR1287 may work its therapeutic effect is by regulating the balance of immune cells. CD4+ T cells are crucial for fighting off infections and cancer, while CD8+ T cells are responsible for killing infected and mutated cells. MIR1287 has been shown to promote the development and activation of CD4+ T cells while inhibiting the activity of CD8+ T cells. This may lead to a reduction in the production of pro-inflammatory cytokines and an improvement in the immune response.

MIR1287 has also been shown to have a positive impact on the expression of genes involved in cell adhesion, migration, and invasion. These genes are important for the development and progression of cancer. MIR1287 has been shown to increase the expression of genes involved in cell adhesion and migration, leading to the formation of cancer cells. This may contribute to the development of cancer.

In addition to its potential therapeutic effects, MIR1287 has also been identified as a potential biomarker for various diseases. The expression of MIR1287 has been shown to be elevated in various tissues and diseases, including cancer, autoimmune diseases, and COPD. This may indicate that MIR1287 could be used as a diagnostic or therapeutic target in these diseases.

MIR1287 has also been shown to have a positive impact on the function of various tissues and organs. For example, MIR1287 has been shown to improve the function of the lungs in individuals with COPD. This may be due to the positive impact of MIR1287 on the production of anti-inflammatory cytokines, which may help to reduce inflammation in the lungs.

In conclusion, MIR1287 is a potential drug target and biomarker for various diseases. Its functions as a regulator of T cell responses and its potential impact on the balance of immune cells, gene expression, and tissue function make it an attractive target for further research. Further studies are needed to fully understand the potential therapeutic and diagnostic uses of MIR1287.

Protein Name: MicroRNA 1287

The "MIR1287 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR1287 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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