Target Name: MIR4492
NCBI ID: G100616376
Review Report on MIR4492 Target / Biomarker Content of Review Report on MIR4492 Target / Biomarker
MIR4492
Other Name(s): microRNA 4492 | MicroRNA 4492 | hsa-mir-4492 | mir-4492 | hsa-miR-4492

MIR4492: A Potential Drug Target and Biomarker for the Treatment of Chronic Pain

Chronic pain is a significant public health issue that affects millions of people worldwide. The pain can be caused by various conditions such as fibromyalgia, osteoarthritis, cancer, and multiple sclerosis, among others. Chronic pain can be a persistent and debilitating condition that significantly affects an individual's quality of life and overall prognosis.

Recently, researchers have discovered that the pain signaling pathway, which involves the use of chemical messengers called neurotransmitters, may play a crucial role in the development and maintenance of chronic pain. MIR4492, a non-coding RNA molecule, has been identified as a potential drug target and biomarker for the treatment of chronic pain.

MIR4492: A Potential Drug Target

MIR4492 is a non-coding RNA molecule that is expressed in various tissues of the body, including the brain, heart, and peripheral tissues. It is a small molecule that has been shown to regulate the activity of several signaling pathways involved in pain signaling.

One of the key functions of MIR4492 is its role in the nociceiton system, which is responsible for the sensation of pain associated with tissue damage or inflammation. MIR4492 has been shown to regulate the activity of nociceiton neurons, which are involved in the perception of pain.

Additionally, MIR4492 has been shown to play a role in the inflammatory response. It has been shown to regulate the production of pro-inflammatory cytokines, which are involved in the recruitment of immune cells to the site of injury or inflammation.

MIR4492 has also been shown to play a role in the regulation of pain modulation. It has been shown to regulate the activity of GABA, which is a neurotransmitter involved in the inhibition of pain.

MIR4492: A Potential Biomarker

In addition to its role as a drug target, MIR4492 has also been shown to be a potential biomarker for the treatment of chronic pain. The development of chronic pain is associated with increased levels of inflammation and hyperactivity in the pain signaling pathway.

MIR4492 has been shown to regulate the activity of pain signaling pathways, which may contribute to the development and maintenance of chronic pain. By targeting MIR4492, researchers may be able to develop new treatments for chronic pain.

Conclusion

Chronic pain is a significant public health issue that can significantly affect an individual's quality of life. The development and maintenance of chronic pain is associated with several signaling pathways, including the nociceiton system, inflammation, and pain modulation.

MIR4492 has been identified as a potential drug target and biomarker for the treatment of chronic pain. Its role in the nociceiton system, inflammation, and pain modulation may make it an attractive target for new treatments for chronic pain. Further research is needed to fully understand the potential of MIR4492 as a drug and biomarker for the treatment of chronic pain.

Protein Name: MicroRNA 4492

The "MIR4492 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR4492 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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