Target Name: MIR4306
NCBI ID: G100422861
Review Report on MIR4306 Target / Biomarker Content of Review Report on MIR4306 Target / Biomarker
MIR4306
Other Name(s): hsa-miR-4306 | microRNA 4306 | MicroRNA 4306 | mir-4306 | hsa-mir-4306

MIR4306: A Potential Drug Target and Biomarker

MIR4306, a gene encoding a protein known as MIR4306, has been identified as a potential drug target and biomarker for various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. Its unique structure and function have made it an attractive target for drug development, with several research groups actively pursuing studies to investigate its potential as a therapeutic agent.

MIR4306 is a protein that is expressed in various tissues and cells throughout the body, including the brain, pancreas, and gastrointestinal tract. It is a member of the Mir gene family, which is known for encoding non-coding RNAs that play critical roles in various cellular processes, including gene regulation, cell survival, and signaling pathways. The MIR4306 gene has been shown to be involved in the regulation of cell proliferation, apoptosis, and inflammation.

One of the key features of MIR4306 is its ability to interact with various signaling pathways, including TGF-β, NF-kappa-B, and PI3K/AKT. TGF-β is a well-known transcription factor that regulates cell growth, differentiation, and apoptosis, while NF-kappa-B is a signaling pathway that regulates inflammation and immune responses. PI3K/AKT is a signaling pathway that is involved in the regulation of cell survival and metabolism. MIR4306 has been shown to play a role in the regulation of these signaling pathways, which suggests that it may be a potential drug target for diseases that are characterized by the disruption of these signaling pathways.

In addition to its potential as a drug target, MIR4306 has also been identified as a potential biomarker for several diseases. The MIR4306 protein is expressed in various tissues and cells, including the brain, which suggests that it may be a potential biomarker for neurological disorders. Several research groups have shown that MIR4306 is overexpressed in various neurological tissues, including the brain, and that it is involved in the regulation of neural development, differentiation, and survival. This suggests that MIR4306 may be a promising biomarker for a variety of neurological disorders, including Alzheimer's disease, Parkinson's disease, and other neurodegenerative disorders.

MIR4306 has also been shown to be involved in the regulation of cancer cell growth and metastasis. Several studies have shown that MIR4306 is overexpressed in various cancer tissues and that it is involved in the regulation of cell proliferation, apoptosis, and angiogenesis. This suggests that MIR4306 may be a potential drug target for cancer therapies that target these signaling pathways.

In conclusion, MIR4306 is a protein that has shown promise as a potential drug target and biomarker for a variety of diseases. Its unique structure and function, as well as its involvement in multiple signaling pathways, make it an attractive target for drug development. Further research is needed to fully understand the mechanisms of MIR4306's function and its potential as a therapeutic agent.

Protein Name: MicroRNA 4306

The "MIR4306 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR4306 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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