Target Name: IFNK
NCBI ID: G56832
Review Report on IFNK Target / Biomarker Content of Review Report on IFNK Target / Biomarker
IFNK
Other Name(s): Interferon kappa | IFNK_HUMAN | INFE1 | IFN-kappa | IFNT1 | interferon-like protein | interferon kappa | RP11-27J8.1 | Interferon-like protein

IFNK: A Potential Drug Target and Biomarker for Interferon-Induced Neoplasia

Interferon is a cytokine that plays a critical role in the immune response against viruses and some diseases. It is a small protein that is derived from the interferon gene, which is located on chromosome 12. Interferon is known for its ability to induce a variety of cellular responses, including the production of activated T cells, the production of monocytes and natural killer cells, and the production of anti-inflammatory cytokines.

One of the well-known functions of interferon is its ability to induce the production of neoplasia, which is the formation of cancerous tumors. Neoplasia is a complex process that involves the development of a variety of different types of cancer, and it is one of the leading causes of cancer-related deaths.

IFNK has been identified as a potential drug target for the treatment of neoplasia. Neoplasia is a disease that is characterized by the formation of tumors, and it can be caused by a variety of different factors, including genetic mutations, infections, and exposure to environmental toxins. Treatment of neoplasia is often difficult and has limited success, and there is a need for new and effective therapies that can effectively treat this disease.

IFNK is a protein that is derived from the interferon gene and has been shown to play a role in the development and progression of neoplasia. Interferon is known for its ability to induce the production of activated T cells, which are a type of immune cell that play a critical role in the immune response against viruses and other pathogens. When T cells are activated, they release a variety of cytokines, including interferon, which can stimulate the production of other immune cells and affect the behavior of the immune system.

IFNK has also been shown to play a role in the regulation of cell proliferation. Neoplasia is characterized by the rapid and uncontrolled growth of cells, and IFNK is known for its ability to regulate the production of signals that promote cell proliferation. When IFNK is activated, it can inhibit the production of signals that promote cell proliferation, which can lead to the inhibition of neoplasia.

IFNK has also been shown to play a role in the regulation of cell apoptosis, which is the process by which cells die naturally. Neoplasia is characterized by the regulation of cell apoptosis, and IFNK is known for its ability to promote apoptosis in cancer cells. This can be an effective way to treat neoplasia, as it can help to kill cancer cells that have become too large and have the potential to form tumors.

IFNK has also been shown to play a role in the regulation of inflammation. Neoplasia is often associated with chronic inflammation, and IFNK is known for its ability to regulate the production of pro-inflammatory cytokines. This can help to contribute to the development and progression of neoplasia.

IFNK has also been shown to play a role in the regulation of the immune response. Neoplasia is often associated with an imbalance in the immune system, and IFNK is known for its ability to regulate the production of immune cells. This can help to contribute to the development and progression of neoplasia.

In conclusion, IFNK is a protein that is derived from the interferon gene and has been shown to play a role in the development and progression of neoplasia. Neoplasia is a complex disease that is characterized by the rapid and uncontrolled growth of cells, and IFNK is known for its ability to regulate the production of signals that promote cell proliferation and the regulation of cell apoptosis,

Protein Name: Interferon Kappa

Functions: May play a role in the regulation of immune cell function. Cytokine that imparts cellular protection against viral infection in a species-specific manner. Activates the interferon-stimulated response element signaling pathway. It is able to directly modulate cytokine release from monocytes and dendritic cells. Binds heparin

The "IFNK Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about IFNK comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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IFNL1 | IFNL2 | IFNL3 | IFNL4 | IFNLR1 | IFNW1 | IFNWP15 | IFNWP19 | IFRD1 | IFRD2 | IFT122 | IFT122P3 | IFT140 | IFT172 | IFT20 | IFT22 | IFT27 | IFT43 | IFT46 | IFT52 | IFT57 | IFT74 | IFT80 | IFT81 | IFT88 | IFTAP | IGBP1 | IGBP1P1 | IGDCC3 | IGDCC4 | IgE Receptors | IGF1 | IGF1R | IGF2 | IGF2-AS | IGF2BP1 | IGF2BP2 | IGF2BP2-AS1 | IGF2BP3 | IGF2R | IGFALS | IGFBP-AS1 | IGFBP1 | IGFBP2 | IGFBP3 | IGFBP4 | IGFBP5 | IGFBP6 | IGFBP7 | IGFBP7-AS1 | IGFBPL1 | IGFL1 | IGFL1P2 | IGFL2 | IGFL2-AS1 | IGFL3 | IGFL4 | IGFLR1 | IGFN1 | IGH@ | IGHA1 | IGHA2 | IGHD | IGHD1-1 | IGHD1-14 | IGHD1-20 | IGHD1-26 | IGHD1-7 | IGHD2-15 | IGHD2-2 | IGHD2-21 | IGHD2-8 | IGHD3-10 | IGHD3-16 | IGHD3-22 | IGHD3-3 | IGHD3-9 | IGHD4-11 | IGHD4-17 | IGHD4-23 | IGHD4-4 | IGHD5-12 | IGHD5-18 | IGHD5-24 | IGHD5-5 | IGHD5OR15-5B | IGHD6-13 | IGHD6-19 | IGHD6-25 | IGHD6-6 | IGHD7-27 | IGHE | IGHEP1 | IGHEP2 | IGHG1 | IGHG2 | IGHG3 | IGHG4 | IGHGP | IGHJ1P