Target Name: IFNA17
NCBI ID: G3451
Review Report on IFNA17 Target / Biomarker Content of Review Report on IFNA17 Target / Biomarker
IFNA17
Other Name(s): interferon alpha-WA | Interferon alpha-17 | interferon alpha 17 | Interferon alpha-I' | LEIF2C1 | leIF I | Interferon alpha-T | IFN17_HUMAN | interferon alpha-I' | IFN-alpha-17 | Interferon alpha-88 |

IFNA17: A Promising Drug Target and Biomarker for the Treatment of Fibrosis

Abstract:

Fibrosis is a widespread disease that affects millions of people worldwide, leading to chronic pain, decreased quality of life, and even death. Interferon alpha-WA (IFNA17) is a drug that has shown great potential in the treatment of fibrosis by targeting the programmed cell death ( apoptosis ) pathway. In this article, we will discuss the science behind IFNA17, its potential as a drug target, and its potential as a biomarker for the diagnosis and monitoring of fibrosis.

Introduction:

Fibrosis is a complex process that involves the activation and proliferation of cells that are damaged or dysfunctional. These cells can promote the growth and progression of fibrotic tissue, leading to the development of chronic diseases such as heart failure, chronic obstructive pulmonary disease (COPD), and progressive systemic joint diseases. The treatment of fibrosis is a major focus of research in the field of medicine, and new treatments are constantly being developed to improve outcomes for patients.

IFNA17: A Drug Target for Fibrosis

IFNA17 is a protein that is expressed in a variety of tissues and cells, including the liver, lung, and heart. It is a member of the interferon family, which includes proteins that play a critical role in the regulation of cellular processes such as cell death and inflammation. IFNA17 has been shown to be involved in the regulation of cell apoptosis, which is the process by which cells undergo programmed cell death.

In fibrosis, the regulation of cell apoptosis is disrupted, leading to the activation and proliferation of damaged cells that promote the growth and progression of fibrotic tissue. IFNA17 has been shown to play a critical role in the regulation of cell apoptosis in fibrotic tissues, by promoting the expression of genes that promote apoptosis and inhibiting the expression of genes that promote cell survival.

IFNA17 has also been shown to be involved in the regulation of angiogenesis, which is the process by which new blood vessels are formed in response to the development of a disease. Angiogenesis is a critical process for the treatment of fibrosis, as the growth and progression of fibrotic tissue is dependent on the availability of oxygen and nutrients. IFNA17 has been shown to play a critical role in the regulation of angiogenesis, by promoting the formation of new blood vessels and the growth of existing blood vessels.

IFNA17 as a Biomarker:

IFNA17 has also been shown to be a potential biomarker for the diagnosis and monitoring of fibrosis. Fibrosis can be diagnosed by the presence of fibrotic tissue in biopsy samples, which can be difficult to obtain and interpret. IFNA17 has been shown to be expressed in the fibrotic tissue, and can be used as a marker for the diagnosis of fibrosis.

IFNA17 has also been shown to be a potential biomarker for the monitoring of fibrosis, as its levels can be easily measured and correlated with the severity of fibrosis. This makes IFNA17 an attractive candidate for use as a marker for the monitoring of fibrosis, particularly in patients with chronic diseases such as COPD and progressive systemic joint diseases.

IFNA17 as a Drug Target:

The potential use of IFNA17 as a drug target for fibrosis is based on its involvement in the regulation of cell apoptosis and angiogenesis. IFNA17 has been shown to play a critical role in the regulation of cell apoptosis, by promoting the expression of genes that promote apoptosis and inhibiting the expression of genes that promote cell survival. This suggests that IF

Protein Name: Interferon Alpha 17

Functions: Produced by macrophages, IFN-alpha have antiviral activities. Interferon stimulates the production of two enzymes: a protein kinase and an oligoadenylate synthetase

The "IFNA17 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about IFNA17 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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IFNA2 | IFNA21 | IFNA22P | IFNA4 | IFNA5 | IFNA6 | IFNA7 | IFNA8 | IFNAR1 | IFNAR2 | IFNB1 | IFNE | IFNG | IFNG-AS1 | IFNGR1 | IFNGR2 | IFNK | IFNL1 | IFNL2 | IFNL3 | IFNL4 | IFNLR1 | IFNW1 | IFNWP15 | IFNWP19 | IFRD1 | IFRD2 | IFT122 | IFT122P3 | IFT140 | IFT172 | IFT20 | IFT22 | IFT27 | IFT43 | IFT46 | IFT52 | IFT57 | IFT74 | IFT80 | IFT81 | IFT88 | IFTAP | IGBP1 | IGBP1P1 | IGDCC3 | IGDCC4 | IgE Receptors | IGF1 | IGF1R | IGF2 | IGF2-AS | IGF2BP1 | IGF2BP2 | IGF2BP2-AS1 | IGF2BP3 | IGF2R | IGFALS | IGFBP-AS1 | IGFBP1 | IGFBP2 | IGFBP3 | IGFBP4 | IGFBP5 | IGFBP6 | IGFBP7 | IGFBP7-AS1 | IGFBPL1 | IGFL1 | IGFL1P2 | IGFL2 | IGFL2-AS1 | IGFL3 | IGFL4 | IGFLR1 | IGFN1 | IGH@ | IGHA1 | IGHA2 | IGHD | IGHD1-1 | IGHD1-14 | IGHD1-20 | IGHD1-26 | IGHD1-7 | IGHD2-15 | IGHD2-2 | IGHD2-21 | IGHD2-8 | IGHD3-10 | IGHD3-16 | IGHD3-22 | IGHD3-3 | IGHD3-9 | IGHD4-11 | IGHD4-17 | IGHD4-23 | IGHD4-4 | IGHD5-12 | IGHD5-18