Target Name: C1QTNF5
NCBI ID: G114902
Review Report on C1QTNF5 Target / Biomarker Content of Review Report on C1QTNF5 Target / Biomarker
C1QTNF5
Other Name(s): LORD | myonectin | C1q TNF-alpha-related protein 5 | C1QTNF5 variant 1 | Complement C1q tumor necrosis factor-related protein 5 | MFRP | Membrane frizzled-related protein | C1q and TNF related 5, transcript variant 1 | C1QT5_HUMAN | CTRP5 | C1q and TNF related 5 | C1q and tumor necrosis factor related protein 5

C1QTNF5: A Protein Implicated in Disease Development

C1QTNF5, also known as Lord, is a protein that is expressed in various tissues throughout the body. It is a member of the C1Q family of proteins, which are known for their role in cell signaling and inflammation. C1QTNF5 has been shown to play a role in the development and progression of various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. As a result, C1QTNF5 has become a focus of interest in the scientific community and a potential drug target.

The C1QTNF5 protein is composed of 218 amino acid residues and has a calculated molecular weight of 23.9 kDa. It is expressed in a variety of tissues, including muscle, nerve, heart, liver, and kidney. C1QTNF5 is highly expressed in the brain and has been shown to be involved in the development and progression of various neurological disorders, including Alzheimer's disease, Parkinson's disease, and neurofibrillary tangles.

In addition to its expression in the brain, C1QTNF5 has also been shown to be involved in the development and progression of other diseases. For example, C1QTNF5 has been shown to be involved in the development of cancer, both in humans and animals. Studies have shown that high levels of C1QTNF5 are associated with an increased risk of cancer and that inhibiting C1QTNF5 may be a potential therapeutic approach for cancer treatment.

Another potential application of C1QTNF5 as a drug target is its role in neurodegenerative diseases. C1QTNF5 has been shown to be involved in the development and progression of various neurodegenerative diseases, including Alzheimer's disease, Parkinson's disease, and Huntington's disease. Studies have shown that reducing C1QTNF5 levels may be a potential therapeutic approach for these diseases.

In addition to its potential role in neurodegenerative diseases, C1QTNF5 has also been shown to be involved in the development and progression of autoimmune disorders. C1QTNF5 has been shown to play a role in the development and progression of autoimmune disorders, including rheumatoid arthritis, lupus, and multiple sclerosis.

C1QTNF5 has also been shown to be involved in various cellular signaling pathways, including the TGF-β pathway. This pathway is involved in cell signaling and is known to play a role in the development and progression of various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders.

In conclusion, C1QTNF5 is a protein that has been shown to play a role in the development and progression of various diseases. As a result, C1QTNF5 has become a focus of interest in the scientific community and a potential drug target. Further research is needed to fully understand the role of C1QTNF5 in disease and to develop effective therapies for its treatment.

Protein Name: C1q And TNF Related 5

The "C1QTNF5 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about C1QTNF5 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

More Common Targets

C1QTNF6 | C1QTNF7 | C1QTNF7-AS1 | C1QTNF8 | C1QTNF9 | C1QTNF9B | C1R | C1RL | C1RL-AS1 | C1S | C2 | C2-AS1 | C20orf141 | C20orf144 | C20orf173 | C20orf181 | C20orf202 | C20orf203 | C20orf204 | C20orf27 | C20orf85 | C20orf96 | C21orf58 | C21orf62 | C21orf62-AS1 | C21orf91 | C21orf91-OT1 | C22orf15 | C22orf23 | C22orf31 | C22orf39 | C22orf42 | C22orf46P | C2CD2 | C2CD2L | C2CD3 | C2CD4A | C2CD4B | C2CD4C | C2CD4D | C2CD4D-AS1 | C2CD5 | C2CD6 | C2orf15 | C2orf16 | C2orf27A | C2orf42 | C2orf48 | C2orf49 | C2orf50 | C2orf66 | C2orf68 | C2orf69 | C2orf72 | C2orf73 | C2orf74 | C2orf74-DT | C2orf76 | C2orf78 | C2orf80 | C2orf81 | C2orf83 | C2orf88 | C2orf92 | C3 | C3AR1 | C3orf14 | C3orf18 | C3orf20 | C3orf22 | C3orf33 | C3orf36 | C3orf38 | C3orf49 | C3orf52 | C3orf62 | C3orf70 | C3orf80 | C3orf84 | C3orf85 | C3orf86P | C3P1 | C4A | C4B | C4BPA | C4BPAP2 | C4BPB | C4B_2 | C4orf17 | C4orf19 | C4orf3 | C4orf33 | C4orf36 | C4orf45 | C4orf46 | C4orf46P3 | C4orf47 | C4orf48 | C4orf50 | C4orf51