IFITM10: A Protein in Many Tissues and Its Implications in Cellular Processes

Target Name: IFITM10

NCBI ID: G402778

IFITM10

IFITM10: A Protein in Many Tissues and Its Implications in Cellular Processes

IFITM10 (DSPA3) is a protein that is expressed in various tissues of the body, including the brain, heart, and kidneys. It is a member of the intermediate filament (IF) family, which is characterized by the presence of a unique transmembrane domain and a cytoplasmic tail. IFITM10 has been shown to play a role in the regulation of various cellular processes, including cell growth, differentiation, and survival.

One of the most significant aspects of IFITM10 is its role in the regulation of cell survival. Many diseases, including cancer, are characterized by the destruction of cells that have become damaged or mutated. IFITM10 has been shown to play a role in the regulation of cell death, either by directly interacting with cell death-related proteins or by contributing to the formation of cell death-related structures.

Another important function of IFITM10 is its role in the regulation of cell growth. Cell growth is an important process that leads to an increase in cell volume and function. IFITM10 plays an important role in cell growth regulation. It can participate in cell growth regulation by directly interacting with cell death-related proteins or by participating in the formation of cell death-related structures.

IFITM10 is also involved in the process of cell differentiation. Cell differentiation is an important process that results in the transformation of cells from an undifferentiated state into specific cell types. IFITM10 plays an important role in the regulation of cell differentiation. It can participate in the regulation of cell differentiation by directly interacting with proteins related to cell differentiation, or by participating in the formation of structures related to cell differentiation.

IFITM10 is also closely related to the occurrence and development of many diseases. Many diseases, including cancer, are associated with dysregulated apoptosis. IFITM10 plays an important role in the regulation of apoptosis. It can participate in the regulation of apoptosis by directly interacting with apoptosis-related proteins or by participating in the formation of apoptosis-related structures.

IFITM10 is also closely related to the morphological characteristics of apoptosis. Morphological characteristics are an important aspect of cell death, which determine the form and degree of cell death. IFITM10 plays an important role in the morphological characteristics of apoptosis. It can participate in the regulation of morphological characteristics of apoptosis by directly interacting with apoptosis-related proteins or by participating in the formation of apoptosis-related structures.

In summary, IFITM10 is a protein that plays an important role in many physiological and pathological processes. Its functions include participating in the regulation of cell growth, differentiation, and apoptosis, so it is considered a potential drug target (or biomarker). In the future, studying the mechanism of action of IFITM10 and exploring its application in drug development will be of great clinical significance.

Protein Name: Interferon Induced Transmembrane Protein 10

The "IFITM10 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about IFITM10 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
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•   the target screening and validation;
•   expression level;
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•   related combination drugs;
•   pharmacochemistry experiments;
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•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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