Target Name: SAA2-SAA4
NCBI ID: G100528017
Review Report on SAA2-SAA4 Target / Biomarker Content of Review Report on SAA2-SAA4 Target / Biomarker
SAA2-SAA4
Other Name(s): SAA2-SAA4 read-through transcript | SAA2-SAA2 read-through transcript | SAA2-SAA2 protein | SAA2-SAA4 readthrough

SAA2-SAA4: A Drug Target / Disease Biomarker

SAA2-SAA4 is a protein that is expressed in various tissues of the body, including the brain, heart, liver, and kidneys. It is a member of the SAA family of proteins, which are known for their role in the regulation of cell signaling pathways. SAA2-SAA4 has been identified as a potential drug target or biomarker for various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders.

The SAA family of proteins are characterized by the presence of a single transmembrane domain and a cytoplasmic tail. They are involved in the regulation of various cellular processes, including cell signaling pathways, cell adhesion, and cell survival. The SAA2-SAA4 protein is a member of this family and is expressed in various tissues of the body.

SAA2-SAA4 is a 21-kDa protein that is expressed in the brain, heart, liver, and kidneys. It is involved in the regulation of various cellular processes, including cell signaling pathways, cell adhesion, and cell survival. SAA2-SAA4 has been shown to play a role in the development and progression of several diseases, including cancer, neurodegenerative diseases, and autoimmune disorders.

One of the key functions of SAA2-SAA4 is its role in cell signaling pathways. SAA2-SAA4 is involved in the regulation of several signaling pathways, including the TGF-β pathway, the PI3K/Akt pathway, and the NF-kappa-B pathway. These pathways are involved in various cellular processes, including cell growth, differentiation, and survival. SAA2-SAA4 has been shown to play a negative role in the regulation of cell growth and survival, which may contribute to its potential as a drug target or biomarker for various diseases.

In addition to its role in cell signaling pathways, SAA2-SAA4 is also involved in the regulation of cell adhesion. SAA2-SAA4 is a member of the cadherin family of proteins, which are involved in cell-cell adhesion. SAA2-SAA4 has been shown to play a role in the regulation of cell-cell adhesion, which is important for the maintenance of tissue structure and function.

SAA2-SAA4 is also involved in the regulation of cell survival. SAA2-SAA4 has been shown to play a role in the regulation of cell survival, including the regulation of apoptosis. SAA2-SAA4 has been shown to interact with several proteins that are involved in cell apoptosis, including Bax and p53. These interactions may contribute to SAA2-SAA4's role in the regulation of cell survival.

In conclusion, SAA2-SAA4 is a protein that is involved in the regulation of various cellular processes, including cell signaling pathways, cell adhesion, and cell survival. Its negative role in the regulation of cell growth and survival may make it a potential drug target or biomarker for various diseases. Further research is needed to fully understand the role of SAA2-SAA4 in the regulation of cellular processes and its potential as a drug target or biomarker.

Protein Name: SAA2-SAA4 Readthrough

The "SAA2-SAA4 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about SAA2-SAA4 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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