Target Name: LGALS7
NCBI ID: G3963
Review Report on LGALS7 Target / Biomarker Content of Review Report on LGALS7 Target / Biomarker
LGALS7
Other Name(s): KL-14 | Lectin galactoside-binding soluble 7 | LGALS7A | PIG1 | galectin 7 | HKL-14 | Galectin 7 | TP53I1 | p53-induced gene 1 protein | lectin, galactoside-binding, soluble, 7 | Gal-7 | Galectin-7 | PI7 | Galectin 7B | LEG7_HUMAN | GAL7

Understanding LGALS7: A Protein Involved in Tissue Growth, Maintenance and Repair

LGALS7 (KL-14) is a protein that is expressed in various tissues throughout the body, including the brain, pancreas, and heart. Its function is not well understood, but it is known to be involved in the regulation of cellular processes that are important for tissue growth, maintenance, and repair.

Recent studies have identified LGALS7 as a potential drug target or biomarker for various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. Its unique structure and subcellular localization make it an attractive target for small molecules, antibodies, or other therapeutic agents that can modulate its activity.

One of the key features of LGALS7 is its unique subcellular localization. Unlike many other proteins, LGALS7 is expressed in all cells of the body and has been shown to be highly enriched in the brain and pancreas, two areas where it is known to play a critical role in the regulation of cellular processes that are important for tissue growth, maintenance, and repair.

This subcellular localization makes LGALS7 an attractive target for small molecules or antibodies that can specifically modulate its activity in these regions. For example, studies have shown that inhibiting the activity of LGALS7 can lead to a variety of cellular and molecular changes, including decreased cell proliferation, increased cell death, and alterations in the expression of other genes.

Another feature of LGALS7 is its role in the regulation of cellular processes that are important for tissue growth and development. LGALS7 has been shown to be involved in the regulation of cell migration, a critical process for the formation of tissues and organs during development.

Additionally, LGALS7 is also involved in the regulation of cellular processes that are important for the maintenance of tissues and organs. Its role in these processes is not well understood, but it is known to be involved in the regulation of angiogenesis, the formation of new blood vessels, and the regulation of blood pressure.

Overall, LGALS7 is a protein that is expressed in various tissues throughout the body and is involved in the regulation of cellular processes that are important for tissue growth, maintenance, and repair. Its unique subcellular localization and involvement in critical cellular processes make it an attractive target for small molecules, antibodies, or other therapeutic agents that can modulate its activity. Further research is needed to fully understand its role in these processes and to develop effective therapies for diseases that are caused by its dysfunction.

Protein Name: Galectin 7

Functions: Could be involved in cell-cell and/or cell-matrix interactions necessary for normal growth control. Pro-apoptotic protein that functions intracellularly upstream of JNK activation and cytochrome c release

The "LGALS7 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about LGALS7 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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