MFSD8: A Potential Drug Target for Neurological Disorders (G256471)

Target Name: MFSD8

NCBI ID: G256471

MFSD8

Other Name(s): Ceroid-lipofuscinosis neuronal protein 7 | ceroid-lipofuscinosis, neuronal 7, late infantile | Ceroid-lipofuscinosis, neuronal 7, late infantile | Major facilitator superfamily domain-containing protein 8 (isoform 1) | CCMD | Major facilitator superfamily domain containing 8, transcript variant 1 | Major facilitator superfamily domain-containing protein 8 | MFSD8_HUMAN | Major facilitator superfamily domain containing 8 | Major facilitator superfamily domain-containing protein 8 isoform 5 | major facilitator superfamily domain containing 8 | CLN7 | MFSD8 variant 1

MFSD8: A Potential Drug Target for Neurological Disorders

Ceroid-lipofuscinosis neuronal protein 7 (MFSD8) is a protein that is expressed in various tissues throughout the body, including the brain. It is a member of the superfamily of reformed development family, which includes other proteins such as Calbindin and S100尾. MFSD8 is known for its unique structure, which consists of a long amino acid sequence that is rich in cysteine 鈥嬧?媟esidues.

One of the unique features of MFSD8 is its ability to interact with other proteins. It has been shown to interact with a wide range of proteins, including neurotransmitter receptors, ion channels, and enzymes. This interaction with other proteins makes MFSD8 an attractive potential drug target.

MFSD8 has also been shown to play a role in the development and progression of various neurological disorders. For example, studies have shown that MFSD8 is expressed in the brains of individuals with Alzheimer's disease, and that it is involved in the progression of the disease. Additionally, MFSD8 has been shown to be involved in the development of Parkinson's disease, and that it may be a potential drug target for this disorder.

Another potential mechanism by which MFSD8 may contribute to the development and progression of neurological disorders is its role in modulating the immune response. MFSD8 has been shown to interact with immune cells and to play a role in regulating the immune response. This interaction with the immune system may contribute to the development and progression of neurological disorders by impairing the immune response and increasing the risk of infection.

In addition to its potential role in the development and progression of neurological disorders, MFSD8 is also a potential biomarker. Studies have shown that MFSD8 can be used as a protein biomarker for various neurological disorders, including Alzheimer's disease, Parkinson's disease, and multiple sclerosis. This makes MFSD8 an attractive potential drug target for these disorders.

Overall, MFSD8 is a protein that has unique properties and functions. Its ability to interact with other proteins and its role in the development and progression of neurological disorders make it an attractive potential drug target. Additionally, MFSD8 is also a potential biomarker for various neurological disorders disorders, which makes it a valuable tool for the development of new treatments.

Protein Name: Major Facilitator Superfamily Domain Containing 8

Functions: May be a carrier that transport small solutes by using chemiosmotic ion gradients

The "MFSD8 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MFSD8 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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