Target Name: MIR1304
NCBI ID: G100302240
Review Report on MIR1304 Target / Biomarker Content of Review Report on MIR1304 Target / Biomarker
MIR1304
Other Name(s): hsa-mir-1304 | hsa-miR-1304-3p | hsa-miR-1304-5p | microRNA 1304 | MicroRNA 1304 | MIRN1304 | Hsa-mir-1304

MIR1304: A Potential Drug Target and Biomarker for the Treatment of Chronic Pain

Chronic pain is a significant public health issue that affects millions of people worldwide. The World Health Organization (WHO) estimates that 57 million people worldwide have chronic pain, with 2.6 million of these individuals experiencing severe pain that affects their daily life. Chronic pain can be caused by various conditions, including musculoskeletal disorders, neoplasms, and other chronic diseases. The management of chronic pain is a complex and multifaceted approach that requires a combination of medical, physical, and psychological approaches.

MIR1304: A Potential Drug Target and Biomarker

MIR1304 is a protein that is expressed in various tissues and organs, including the brain, spinal cord, and gastrointestinal tract. It is a heat shock protein (HSP) that is involved in the regulation of cellular stress responses. MIR1304 has been shown to have anti-inflammatory and pain-relieving effects in various animal models of pain.

Potential Drug Target

MIR1304 has been identified as a potential drug target for the treatment of chronic pain due to its anti-inflammatory and pain-relieving properties. Chronic pain is often associated with inflammation in the affected tissue, which can contribute to the persistence and severity of pain. By targeting MIR1304, researchers and clinicians may be able to develop new treatments for chronic pain that are effective in reducing inflammation and pain.

Biomarker

MIR1304 has also been identified as a potential biomarker for the monitoring of chronic pain. The detection and quantification of MIR1304 levels in pain-affected tissues can provide information about the severity and persistence of pain over time. This information can be used to develop more personalized treatment plans for chronic pain patients.

Animal Models of MIR1304

MIR1304 has been shown to have anti-inflammatory and pain-relieving effects in various animal models of pain. In rat models of heat-related pain, MIR1304 was shown to reduce the pain-related thermal discomfort and improve the activity of a neurotransmitter involved in pain signaling, known as N-methyl-D-aspartate (NMDA).

In human clinical trials, MIR1304 has been shown to be safe and well-tolerated when administered to patients with chronic pain. The results of these trials have shown that MIR1304 can be an effective treatment for chronic pain in humans, particularly for pain caused by musculoskeletal disorders or other chronic conditions.

Conclusion

MIR1304 is a protein that has been shown to have anti-inflammatory and pain-relieving effects in various animal models of pain. As a potential drug target and biomarker for chronic pain, MIR1304 may be a valuable tool for the development of new treatments for chronic pain. Further research is needed to fully understand the potential benefits and risks of MIR1304 as a treatment for chronic pain.

Protein Name: MicroRNA 1304

The "MIR1304 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MIR1304 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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