Unlocking the Potential of F11R: A Promising Drug Target and Biomarker for platelet Adhesion Molecule 1

Target Name: F11R

NCBI ID: G50848

F11R

Unlocking the Potential of F11R: A Promising Drug Target and Biomarker for platelet Adhesion Molecule 1

Platelet adhesion molecule 1 (F11R) is a protein that plays a critical role in the regulation of platelet functions, including blood clotting and wound healing. F11R has been identified as a potential drug target and biomarker for various diseases, making it an attractive target for drug development. In this article, we will explore the biology of F11R, its functions, and its potential as a drug target and biomarker.

F11R: A Critical Protein for Platelet Function

Platelets are essential for several functions in the body, including blood clotting, which helps to prevent blood loss in the event of an injury or illness. Platelets also play a critical role in wound healing, where they help to promote the formation of new tissues and close off wounds. F11R is a protein that is expressed in high levels in platelets and is involved in the regulation of several platelet functions.

F11R functions as a negative regulator of the F11R-Fc domain, which is a critical region of the protein that interacts with other proteins. The F11R-Fc domain is responsible for several functions, including the regulation of platelet aggregation, clot retraction, and the ability to form stable, monolayer platelets. F11R plays a critical role in the regulation of these functions by interacting with several other proteins, including F11R-interactive proteins and F11R-associated proteins.

F11R-Induced Platelet Aggregation and Clot Formation

F11R has been shown to play a critical role in the regulation of platelet aggregation and clot formation. In several studies, researchers have shown that inhibition of F11R can prevent platelet aggregation and clot formation, leading to the prevention of blood loss and other potential therapeutic applications.

One of the most significant functions of F11R is its ability to regulate platelet aggregation. Platelet aggregation is a critical step in the pathology of many diseases, including heart disease and cancer. F11R has been shown to play a critical role in regulating platelet aggregation by interacting with F11R-interactive proteins, which are involved in the regulation of platelet functions.

Another function of F11R is its ability to regulate clot formation. Clot formation is a critical step in the pathology of many diseases, including heart disease and cancer. F11R has been shown to play a critical role in regulating clot formation by interacting with F11R-associated proteins, which are involved in the regulation of platelet functions.

F11R as a Potential Drug Target

F11R has been identified as a potential drug target for several diseases due to its critical role in the regulation of platelet functions. Drugs that can inhibit F11R have been shown to have therapeutic applications in a variety of diseases, including heart disease, cancer, and wound healing.

One of the most promising potential drugs that can inhibit F11R is the small molecule inhibitor, F11R-1. F11R-1 has been shown to inhibit the activity of F11R and prevent platelet aggregation and clot formation. Studies have shown that F11R-1 can be effective in preventing blood clots and reducing the risk of heart disease in animal models of disease.

Another potential drug that can inhibit F11R is the monoclonal antibody, F11R-2. F11R-2 has been shown to be highly effective in blocking the activity of F11R and preventing plate

Protein Name: F11 Receptor

Functions: Seems to play a role in epithelial tight junction formation. Appears early in primordial forms of cell junctions and recruits PARD3 (PubMed:11489913). The association of the PARD6-PARD3 complex may prevent the interaction of PARD3 with JAM1, thereby preventing tight junction assembly (By similarity). Plays a role in regulating monocyte transmigration involved in integrity of epithelial barrier (By similarity). Ligand for integrin alpha-L/beta-2 involved in memory T-cell and neutrophil transmigration (PubMed:11812992). Involved in platelet activation (PubMed:10753840)

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The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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