Target Name: OR6C65
NCBI ID: G403282
Review Report on OR6C65 Target / Biomarker Content of Review Report on OR6C65 Target / Biomarker
OR6C65
Other Name(s): O6C65_HUMAN | olfactory receptor family 6 subfamily C member 65 | Olfactory receptor 6C65 | Olfactory receptor family 6 subfamily C member 65

OR6C65: A Potential Drug Target and Biomarker for Diseases

OR6C65 (O6C65_HUMAN), a protein encoded in the human gene O6C65, is a potential drug target and biomarker for various diseases, including cancer, neurodegenerative disorders, and autoimmune diseases. OR6C65 is a key regulator of the Notch signaling pathway, a well-established pathway involved in cell proliferation, survival, and development. The Notch signaling pathway has been implicated in the development and progression of numerous diseases, including cancer, neurodegenerative disorders, and autoimmune diseases.

The Notch signaling pathway is composed of several transcription factors, including Notch1, Notch2, Notch3, and Notch4. These transcription factors are involved in the regulation of gene expression and are responsible for the formation of splicing response (Notch-induced gene expression) in response to Notch signaling. OR6C65 is a key regulator of the Notch signaling pathway, as it is involved in the formation of shear response in response to Notch signaling.

OR6C65 plays a crucial role in the regulation of stem cell proliferation and differentiation. Stem cells are a diverse group of cells that have the ability to self-renew and differentiate into various cell types in the body. a critical process in the development and maintenance of tissues and organs. OR6C65 is involved in the regulation of stem cell proliferation by promoting the formation of shear response, which is a critical signaling pathway involved in stem cell self-renewal and differentiation.

OR6C65 is also involved in the regulation of neurodegenerative disorders, including Alzheimer's disease, Parkinson's disease, and Huntington's disease. These disorders are characterized by the progressive loss of neurons and the disruption of normal brain function. OR6C65 is involved in the regulation of neurodegenerative disorders by preventing the formation of aggregates of the protein N-cadherin, which is involved in the regulation of neural stem cells and the development of neural circuits.

OR6C65 is also a potential biomarker for cancer, as it has been shown to be expressed in various types of cancer. The expression of OR6C65 in cancer cells may be a sign of their aggressive and malignant behavior. OR6C65 has also been shown to be involved in the regulation of cancer cell migration, which is a critical process in the development of cancer.

In conclusion, OR6C65 is a protein that is involved in the regulation of several critical processes in the development and maintenance of tissues and organs, including stem cell proliferation and differentiation, neurodegenerative disorders, and cancer. Its involvement in these processes makes it a potential drug target and biomarker for various diseases. Further research is needed to fully understand the role of OR6C65 in these processes and to develop effective therapies for these diseases.

Protein Name: Olfactory Receptor Family 6 Subfamily C Member 65

Functions: Odorant receptor

The "OR6C65 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about OR6C65 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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