Target Name: AAGAB
NCBI ID: G79719
Review Report on AAGAB Target / Biomarker Content of Review Report on AAGAB Target / Biomarker
AAGAB
Other Name(s): p34 | Alpha- and gamma-adaptin-binding protein p34 | AAGAB_HUMAN | P34 | FLJ11506 | AAGAB variant 1 | Alpha- and gamma-adaptin-binding protein p34 (isoform 1) | Alpha and gamma adaptin binding protein, transcript variant 1 | PPKP1 | PPKP1A | KPPP1 | alpha and gamma adaptin binding protein

AGAB: A Potential Drug Target and Biomarker

The article \"AGAB: A Potential Drug Target and Biomarker\" discusses the molecule AGB-1212, also known as AAGAB (p34), and its potential as a drug target and biomarker. The AGB-1212 molecule is a small protein that is expressed in various tissues and cells in the body, including the brain, heart, and kidneys. It is composed of 121 amino acid residues and has a calculated pI of 9.6.

TheAGAB molecule is involved in various physiological processes in the body, including inflammation, neurodegeneration, and autophagy. It has been shown to play a role in the regulation of cellular processes that are important for brain health and function, such as the regulation of neurotransmitter release, neuroprotection, and stress resistance.

TheAGAB molecule has also been shown to be involved in the development and progression of various diseases, including neurodegenerative diseases, such as Alzheimer's disease, Parkinson's disease, and Huntington's disease. It has been shown to be involved in the regulation of neurotransmitter release from neurons, which is important for the transmission of signals within the brain. It has also been shown to play a role in the regulation of inflammation, which is important for the immune response and the regulation of cellular processes that are important for brain health and function.

TheAGAB molecule has also been shown to be a potential biomarker for various diseases, including neurodegenerative diseases. It has been shown to be involved in the regulation of neurotransmitter release from neurons, which is important for the transmission of signals within the brain. It has also been shown to play a role in the regulation of inflammation, which is important for the immune response and the regulation of cellular processes that are important for brain health and function.

In conclusion, the AGB-1212 molecule, also known as AAGAB (p34), is a potential drug target and biomarker. Its involvement in various physiological processes in the body, including inflammation, neurodegeneration, and autophagy, makes it an attractive target for drug development. Its potential as a drug target is based on its involvement in the regulation of cellular processes that are important for brain health and function, as well as its involvement in the regulation of neurotransmitter release and inflammation. As a biomarker, its potential is based on its involvement in the regulation of neurotransmitter release and its association with the development and progression of various diseases, including neurodegenerative diseases. Further research is needed to fully understand the potential of AGB-1212 as a drug target and biomarker.

Protein Name: Alpha And Gamma Adaptin Binding Protein

Functions: May be involved in endocytic recycling of growth factor receptors such as EGFR

The "AAGAB Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about AAGAB comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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