Target Name: MFAP3L
NCBI ID: G9848
Review Report on MFAP3L Target / Biomarker Content of Review Report on MFAP3L Target / Biomarker
MFAP3L
Other Name(s): OTTHUMP00000219394 | Microfibrillar-associated protein 3-like | MFAP3L variant 1 | OTTHUMP00000219393 | microfibrillar associated protein 3 like | testis development protein NYD-SP9 | OTTHUMP00000219390 | microfibril associated protein 3 like | MFA3L_HUMAN | NYD-sp9 | Testis development protein NYD-SP9 | Microfibrillar-associated protein 3-like (isoform 1) | microfi brillar-associated protein 3-like | Microfibril associated protein 3 like, transcript variant 1 | KIAA0626

MFAP3L: A Potential Drug Target and Biomarker

MFAP3L (Mesothelin-associated protein 3-like) is a protein that is expressed in various tissues and cell types in the human body. It is a member of the mesothelin family, which is known for its role in cell-cell adhesion and invasion. MFAP3L has been identified as a potential drug target and biomarker for various diseases, including cancer, neurodegenerative disorders, and autoimmune diseases.

MFAP3L is expressed in various tissues, including the brain, pancreas, and skin, and has been shown to play a role in several biological processes, including cell adhesion, migration, and invasion. It is a strong candidate for drug targeting due to its unique structure and its involvement in several disease processes.

One of the key features of MFAP3L is its ability to interact with various signaling pathways. MFAP3L has been shown to interact with several signaling pathways, including the TGF-β pathway, the PI3K/Akt pathway, and the NF-kappa-B pathway. These interactions make MFAP3L a potentially interesting drug target for diseases that are caused by disruptions in these signaling pathways.

MFAP3L has also been shown to play a role in several diseases, including cancer, neurodegenerative disorders, and autoimmune diseases. For example, MFAP3L has been shown to be overexpressed in several types of cancer, including breast, ovarian, and prostate cancer. This suggests that MFAP3L may be a useful biomarker for these diseases and may also be a potential drug target.

In addition to its potential clinical applications, MFAP3L is also a potential biomarker for several diseases. The expression of MFAP3L has been shown to be elevated in several types of neurodegenerative disorders, including Alzheimer's disease and Parkinson's disease. This suggests that MFAP3L may be a useful biomarker for these disorders and may also be a potential drug target.

MFAP3L has also been shown to be involved in several autoimmune diseases, including rheumatoid arthritis and multiple sclerosis. This suggests that MFAP3L may be a useful biomarker for these diseases and may also be a potential drug target.

In conclusion, MFAP3L is a protein that has been identified as a potential drug target and biomarker for several diseases. Its unique structure and its involvement in several biological processes make it an attractive target for drug development. Further research is needed to fully understand the role of MFAP3L in disease and to develop effective treatments.

Protein Name: Microfibril Associated Protein 3 Like

Functions: May participate in the nuclear signaling of EGFR and MAPK1/ERK2. May a have a role in metastasis

The "MFAP3L Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MFAP3L comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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