Target Name: DNTT
NCBI ID: G1791
Review Report on DNTT Target / Biomarker Content of Review Report on DNTT Target / Biomarker
DNTT
Other Name(s): DNA nucleotidylexotransferase | Terminal deoxynucleotidyltransferase | terminal transferase | Terminal deoxyribonucleotidyltransferase | OTTHUMP00000020171 | Nucleosidetriphosphate:DNA deoxynucleotidylexotransferase | Deoxyribonucleic nucleotidyltransferase | DNA nucleotidylexotransferase, transcript variant 1 | TDT | terminal deoxynucleotidyltransferase | Terminal addition enzyme | nucleosidetriphosphate:DNA deoxynucleotidylexotransferase | DNA nucleotidylexotransferase (isoform 1) | Deoxyribonucleic acid nucleotidyltransferase | Deoxynucleotidyl terminal transferase | Terminal transferase | terminal deoxyribonucleotidyltransferase | terminal addition enzyme | Addase | Terminal deoxynucleotide transferase | TdT | DNTT variant 1 | TDT_HUMAN

DNTT: A Drug Target / Disease Biomarker

DNTT, short for double-negative transistor, is a technology that has the potential to revolutionize the way we conduct electronic interviews. Unlike traditional transistors, which have a positive and negative polarity, DNTT uses a double-negative approach, where both positive and negative terminals are used to control the flow of current. This allows for more efficient and reliable control of the transistor, and can lead to significant improvements in performance.

DNTT technology has the potential to be a drug target, as it can be used to treat a variety of conditions that involve the manipulation of electrical signals in the brain. For example, DNTT has been shown to be effective in treating conditions such as epilepsy, bipolar disorder, and even addiction, by altering the activity of certain neural pathways in the brain.

In addition to its potential as a drug, DNTT also has great potential as a biomarker. The technology is non-invasive, which means it does not require any incisions or injections, making it a convenient and easy-to-use tool for researchers to study the effects of drugs on brain activity. Additionally, DNTT is highly sensitive, which allows for the detection of small changes in neural activity that may be difficult to detect with other methods.

DNTT has also been shown to be effective in treating certain neurological disorders, such as multiple sclerosis and Parkinson's disease, by reducing the activity of toxic proteins in the brain. Additionally, it has the potential to be used in the development of new treatments for addiction, as it can be used to reduce the cravings for drugs.

Despite the potential benefits of DNTT, there are also concerns about its potential drawbacks. One of the main concerns is the potential for DNTT to cause harm, such as muscle weakness or paralysis, particularly if it is used to stimulate the production of certain proteins. Additionally, there are concerns about the long-term effects of using DNTT, particularly if it is used to treat conditions that involve long-term brain activity.

Despite these concerns, DNTT has the potential to be a valuable tool for the treatment of a variety of neurological disorders. Its ability to control the flow of current, as well as its non-invasive and sensitive nature, make it an attractive option for researchers and doctors looking for new treatments for a range of conditions. As the field of neuroscience continues to evolve, it will be interesting to see how DNTT technology develops and how it may be used in the future.

Protein Name: DNA Nucleotidylexotransferase

Functions: Template-independent DNA polymerase which catalyzes the random addition of deoxynucleoside 5'-triphosphate to the 3'-end of a DNA initiator. One of the in vivo functions of this enzyme is the addition of nucleotides at the junction (N region) of rearranged Ig heavy chain and T-cell receptor gene segments during the maturation of B- and T-cells

The "DNTT Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about DNTT comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

More Common Targets

DNTTIP1 | DNTTIP2 | DOC2A | DOC2B | DOC2GP | DOCK1 | DOCK10 | DOCK11 | DOCK2 | DOCK3 | DOCK4 | DOCK4-AS1 | DOCK5 | DOCK6 | DOCK7 | DOCK8 | DOCK8-AS1 | DOCK9 | DOCK9-DT | DOHH | DOK1 | DOK2 | DOK3 | DOK4 | DOK5 | DOK6 | DOK7 | Dolichol-phosphate-mannose synthase complex | DOLK | DOLPP1 | DONSON | DOP1A | DOP1B | Dopamine receptor | DOT1L | Double homeobox protein 4 | DP2-E2F4 complex | DPAGT1 | DPCD | DPEP1 | DPEP2 | DPEP3 | DPF1 | DPF2 | DPF3 | DPH1 | DPH2 | DPH3 | DPH3P1 | DPH5 | DPH5-DT | DPH6 | DPH6-DT | DPH7 | DPM1 | DPM2 | DPM3 | DPP10 | DPP10-AS1 | DPP3 | DPP3-DT | DPP4 | DPP6 | DPP7 | DPP8 | DPP9 | DPP9-AS1 | DPPA2 | DPPA2P3 | DPPA3 | DPPA3P1 | DPPA3P2 | DPPA4 | DPPA4P3 | DPPA5 | DPPA5P4 | DPRX | DPRXP2 | DPRXP4 | DPT | DPY19L1 | DPY19L1P1 | DPY19L2 | DPY19L2P1 | DPY19L2P2 | DPY19L2P3 | DPY19L2P4 | DPY19L3 | DPY19L3-DT | DPY19L4 | DPY30 | DPYD | DPYD-AS1 | DPYS | DPYSL2 | DPYSL3 | DPYSL4 | DPYSL5 | DQX1 | DR1