Target Name: KCNK10
NCBI ID: G54207
Review Report on KCNK10 Target / Biomarker Content of Review Report on KCNK10 Target / Biomarker
KCNK10
Other Name(s): Potassium two pore domain channel subfamily K member 10, transcript variant 1 | Potassium channel subfamily K member 10 | K2p10.1 | KCNK10 variant 1 | potassium two pore domain channel subfamily K member 10 | TWIK-related K+ channel 2 | potassium channel TREK-2 | potassium channel, subfamily K, member 10 | Potassium channel subfamily K member 10 (isoform 1) | potassium channel, two pore domain subfamily K, member 10 | Outward rectifying potassium channel protein TREK-2 | PPP1R97 | outward rectifying potassium channel protein TREK-2 | TREK-2 | TREK2 | protein phosphatase 1, regulatory subunit 97 | 2P domain potassium channel TREK2 | TREK-2 K(+) channel subunit | KCNKA_HUMAN | Potassium channel TREK-2

KCNK10: A Drug Target / Disease Biomarker

KCNK10, also known as hsp70, is a protein that is expressed in a variety of tissues throughout the body. It is a heat shock protein (HSP) that is involved in a variety of cellular processes, including stress response, DNA damage repair, and cell signaling.

One of the key functions of KCNK10 is its role in the DNA damage repair pathway. This pathway is important for ensuring that damaged DNA is repaired and restored to its original state, which is crucial for the proper functioning of cells. When DNA is damaged or repaired, the cell needs to be able to quickly and efficiently respond to these changes in order to maintain the integrity of its genetic material.

KCNK10 plays a key role in this process by helping to regulate the activity of a protein called DNA damage-inducible gene 1 (DDI-1). This protein is involved in the repair of DNA damage, and it is thought to play a role in the development of certain types of cancer.

Another function of KCNK10 is its role in cell signaling. Many proteins are involved in signaling pathways that help cells communicate with one another and coordinate their activities. KCNK10 is also involved in these signaling pathways, and it is thought to play a role in the regulation of cell proliferation.

In addition to its role in the DNA damage repair pathway and cell signaling, KCNK10 is also involved in the regulation of inflammation. Many diseases, including cancer, are caused by chronic inflammation. KCNK10 is thought to play a role in the regulation of this inflammation, and it is a potential drug target for the treatment of these diseases.

Despite the many important functions of KCNK10, it is not yet clear how it can be used to treat disease. However, research into its potential drug targets and biomarkers is ongoing.

In conclusion, KCNK10 is a protein that is involved in a variety of cellular processes throughout the body. It is a HSP that is involved in the DNA damage repair pathway, cell signaling, and inflammation. While it is not yet clear how it can be used to treat disease, research into its potential drug targets and biomarkers is ongoing. Further studies are needed to fully understand its functions and potential uses in medicine.

Protein Name: Potassium Two Pore Domain Channel Subfamily K Member 10

Functions: Outward rectifying potassium channel. Produces rapidly activating and non-inactivating outward rectifier K(+) currents. Activated by arachidonic acid and other naturally occurring unsaturated free fatty acids

The "KCNK10 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about KCNK10 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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