Target Name: CGB5
NCBI ID: G93659
Review Report on CGB5 Target / Biomarker Content of Review Report on CGB5 Target / Biomarker
CGB5
Other Name(s): hCGB | chorionic gonadotropin subunit beta 5 | CGB7 | Chorionic gonadotrophin chain beta | CGB3_HUMAN | chorionic gonadotropin beta subunit 5 | CGB8 | Chorionic gonadotropin, beta polypeptide 5 | MGC119822 | Choriogonadotropin subunit beta 3 | Chorionic gonadotropin beta 5 subunit | HCG | CGB3 | Chorionic gonadotropin chain beta | chorionic gonadotropin beta 5 subunit | Choriogonadotropin subunit beta | Chorionic gonadotropin subunit beta 5 | CG-beta | CGB

CGB5: Potential Drug Target and Biomarker

CGB5, also known as hCGB, is a protein that is expressed in various tissues throughout the body, including the brain, heart, and kidneys. It is a potential drug target and has been shown to play a role in a number of biological processes, including inflammation, fibrosis, and neurodegeneration.

One of the key functions of CGB5 is its role in the regulation of inflammation. CGB5 has been shown to play a key role in the production of pro-inflammatory cytokines, such as TNF-alpha, IL-1beta, and IL-6. These cytokines can contribute to the inflammatory response that occurs in response to injury, infection, or other stimuli.

In addition to its role in inflammation, CGB5 has also been shown to be involved in the regulation of fibrosis. Fibrosis is the process by which cells in the body respond to injury or damage by causing them to grow and divide in an uncontrolled manner. This process can lead to the development of tumors, chronic pain, and other problems.

Another potential function of CGB5 is its role in neurodegeneration. Neurodegeneration is the process by which the brain and other nervous cells are destroyed or damaged over time. This process can be caused by a variety of factors, including neurotrauma, genetic mutations, and aging.

In addition to its potential functions in inflammation, fibrosis, and neurodegeneration, CGB5 has also been shown to play a role in the regulation of cellular processes that are important for brain function. For example, studies have shown that CGB5 can interact with a variety of brain-derived neurotrophic factor (BDNF) proteins, which are important for the survival and growth of brain cells.

Overall, CGB5 is a protein that has the potential to be a drug target or biomarker for a variety of conditions. Its role in the regulation of inflammation, fibrosis, and neurodegeneration, as well as its interaction with brain-derived neurotrophic factor (BDNF) proteins, make it a promising target for further research.

Protein Name: Chorionic Gonadotropin Subunit Beta 5

Functions: Beta subunit of the human chorionic gonadotropin (hCG). hCG is a complex glycoprotein composed of two glycosylated subunits alpha and beta which are non-covalently associated. The alpha subunit is identical to those in the pituitary gonadotropin hormones (LH, FSH and TSH). The beta subunits are distinct in each of the hormones and confer receptor and biological specificity. Has an essential role in pregnancy and maternal adaptation. Stimulates the ovaries to synthesize the steroids that are essential for the maintenance of pregnancy

The "CGB5 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about CGB5 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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