Target Name: C7orf13
NCBI ID: G129790
Review Report on C7orf13 Target / Biomarker Content of Review Report on C7orf13 Target / Biomarker
C7orf13
Other Name(s): Chromosome 7 open reading frame 13 | MY040

Unveiling the Potential Drug Target and Biomarker C7orf13 in Chromosome 7 Open Reading Frame 13

Introduction

Chromosome 7 (7p) is one of the chromosomes that contribute to the genetic diversity of humans. It is home to numerous genes, including the open reading frame (ORF) 13 gene, which has been identified as a potential drug target and biomarker. In this article, we will delve into the research on C7orf13, exploring its potential as a drug target and biomarker in Chromosome 7 open reading frame 13.

C7orf13: A Potential Drug Target

The C7ORF13 gene is located on chromosome 7p11.2 and encodes a protein known as C7ORF13. This gene has been implicated in the development and progression of various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders.

Recent studies have suggested that C7ORF13 may serve as a potential drug target by regulating the expression of genes involved in cell growth, differentiation, and survival. For instance, overexpression of the C7ORF13 gene has been shown to promote the growth and survival of cancer cells, while inhibition has been shown to negatively affect cell proliferation in neurodegenerative diseases.

Additionally, C7ORF13 has been associated with the expression of genes involved in the immune response, suggesting that it may play a role in the regulation of autoimmune disorders.

C7orf13 as a Biomarker

The discovery of potential drug targets is often accompanied by the identification of biomarkers, which are molecules that can be used as indicators of disease status or response to treatment. In the case of C7ORF13, its potential as a biomarker has been evaluated in various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders.

In cancer, C7ORF13 has been shown to be involved in the regulation of cell growth, differentiation, and survival, which may make it an attractive target for small molecules or antibodies that can inhibit its activity. Several studies have shown that inhibition of C7ORF13 can lead to the regression of cancer tumors in both human and animal models.

In neurodegenerative diseases, C7ORF13 has been associated with the expression of genes involved in the immune response and inflammation. Therefore, targeting C7ORF13 with small molecules or antibodies that can modulate its activity may be a promising approach to treating neurodegenerative diseases.

In autoimmune disorders, C7ORF13 has been shown to be involved in the regulation of immune response genes. Therefore, inhibition of C7ORF13 activity using small molecules or antibodies may be a promising approach to treating autoimmune disorders.

Conclusion

In conclusion, C7ORF13 is a gene that has been identified as a potential drug target and biomarker in Chromosome 7 open reading frame 13. Its potential as a drug target has been evaluated in various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. Further research is needed to fully understand the implications of C7ORF13 as a drug target and biomarker.

Protein Name: Chromosome 7 Open Reading Frame 13

The "C7orf13 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about C7orf13 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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