IL2RA: A Protein Involved in Immune, Inflammatory and Cell Survival

Target Name: IL2RA

NCBI ID: G3559

IL2RA

IL2RA: A Protein Involved in Immune, Inflammatory and Cell Survival

IL2RA (Interleukin 2 Receptor Subunit Alpha) is a protein that is expressed in various tissues throughout the body, including the immune system, skin, and tissues. It is a subunit of the interleukin 2 receptor, which is a protein that is involved in the regulation of immune responses and inflammation.

IL2RA is a type of transmembrane protein that is characterized by its extracellular domain, which is involved in cell-cell signaling. It consists of four constant and one variable region. The constant regions are involved in the formation of the protein's transmembrane domain, while the variable region is involved in the formation of the protein's cytoplasmic domain.

IL2RA is involved in the regulation of various cellular processes, including immune responses, inflammation, and cell survival. It is also involved in the regulation of cellular signaling pathways, including the T cell signaling pathway and the cell survival signaling pathway.

One of the functions of IL2RA is its role in the regulation of T cell responses. T cells are a type of immune cell that play a critical role in the regulation of immune responses and inflammation. IL2RA is involved in the regulation of T cell responses by interacting with the T cell receptor, which is a protein that is involved in the regulation of T cell responses.

IL2RA is also involved in the regulation of inflammation. It is involved in the regulation of the production of pro-inflammatory cytokines, such as interleukin-1尾 and interleukin-6, by interacting with the nuclear factor kappa B (NFKB) signaling pathway.

IL2RA is also involved in the regulation of cell survival. It is involved in the regulation of cell cycle progression by interacting with the cyclin D1 (CDK4) protein. CDK4 is a protein that is involved in the regulation of cell cycle progression, and IL2RA is involved in the regulation of its activity by interacting with it.

Due to its involvement in various cellular processes, IL2RA is a potential drug target. Researchers are currently working to develop drugs that can inhibit the activity of IL2RA in order to treat various diseases, including cancer, autoimmune diseases, and respiratory infections.

In addition to its potential as a drug target, IL2RA is also a potential biomarker. Researchers are currently working to develop methods for the detection and quantification of IL2RA in various tissues and fluids, including blood, urine, and tissue samples. This research has the potential to provide valuable information about the biology of IL2RA and its role in various diseases.

Overall, IL2RA is a protein that is involved in the regulation of various cellular processes, including immune responses, inflammation, and cell survival. It is a potential drug target and biomarker, and research is currently being conducted to further understand its role in these processes.

Protein Name: Interleukin 2 Receptor Subunit Alpha

Functions: Receptor for interleukin-2. The receptor is involved in the regulation of immune tolerance by controlling regulatory T cells (TREGs) activity. TREGs suppress the activation and expansion of autoreactive T-cells

The "IL2RA Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about IL2RA comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
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•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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