Target Name: NTM
NCBI ID: G50863
Review Report on NTM Target / Biomarker Content of Review Report on NTM Target / Biomarker
NTM
Other Name(s): Neurotrimin (isoform 2) | MGC60329 | Neurotrimin (isoform 4) | NT | NTRI_HUMAN | OTTHUMP00000069165 | Neurotrimin, transcript variant 4 | Neurotrimin | neurotrimin | IGLON2 | NTRI | Neurotrimin (isoform 3) | Neurotrimin [Precursor] | HNT | CEPU-1 | hNT | Neurotrimin, transcript variant 3 | NTM variant 2 | OTTHUMP00000069166 | NTM variant 4 | IgLON family member 2 | Neurotrimin, transcript variant 2 | NTM variant 3

Trimin: A Potent Drug Target and Biomarker for Neurological Disorders

Neurotrimin (ISO Form 2) is a drug target and a biomarker that has been gaining significant attention in recent years due to its potential role in the treatment of various neurological disorders. Trimin is a small molecule that is derived from the neurotransmitter neurotrophin (NT) and has been shown to play a critical role in the regulation of neural circuits and behaviors.

One of the key features of Trimin is its ability to modulate the activity of NT-receptors, which are a family of G protein-coupled receptors that play a central role in the regulation of diverse physiological processes, including neuronal function and survival. These receptors are found throughout the central nervous system and play a crucial role in the development and maintenance of normal brain function.

In addition to its ability to modulate NT-receptors, Trimin has also been shown to have a number of other unique properties that make it an attractive drug target. For example, Trimin is highly specific for its interactions with these receptors, meaning that it has a low probability of binding to other types of receptors. This allows for a high degree of selectivity and minimizes the potential for unintended side effects.

Another key feature of Trimin is its ability to modulate the activity of these receptors in a dose-dependent manner. This allows for the precise dosing regimen to be established and provides a more controlled approach to drug treatment. Additionally, Trimin has been shown to have a long half-life, which allows for it to be administered once orally for several days without the need for multiple doses.

In addition to its potential therapeutic applications, Trimin has also been shown to have a number of potential biomarkers. For example, Trimin has been shown to increase the amount of neurotrophin released by NT-receptors in response toNT-agonizing substances, which could be used as a potential diagnostic marker for neurodegenerative diseases. Additionally, the levels of Trimin have been shown to be reduced in individuals with certain neurological disorders, which could be used as a potential biomarker for these disorders.

Overall, Neurotrimin (ISO Form 2) is a drug target and biomarker that has a high degree of potential for the treatment of various neurological disorders. Its ability to modulate NT-receptors and its dose-dependent, dose-independent effects make it an attractive candidate for drug development. Additionally, its ability to serve as a potential biomarker for a number of neurological disorders makes it an important tool for the study of these conditions.

Protein Name: Neurotrimin

Functions: Neural cell adhesion molecule

The "NTM Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about NTM comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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