Target Name: MAML3
NCBI ID: G55534
Review Report on MAML3 Target / Biomarker Content of Review Report on MAML3 Target / Biomarker
MAML3
Other Name(s): CAGH3 | Mastermind-like 3 | polyglutamine rich | KIAA1816 | MAM-2 | CAG repeat containing (glia-derived nexin I alpha) | expanded repeat domain, CAG/CTG 3 | Mam-3 | GDN | Polyglutamine rich | mam-3 | Mastermind-like protein 3 | trinucleotide repeat containing 3 | MAML3_HUMAN | Trinucleotide repeat containing 3 | Expanded repeat domain, CAG/CTG 3 | MAM2 | mastermind like transcriptional coactivator 3 | TNRC3 | Mastermind-like 3 (Drosophila) | CAG repeat domain | ERDA3

MAML3: A Potential Drug Target Or Biomarker

MAML3 (Mammalian APR library gene 3) is a gene that has been identified as a potential drug target or biomarker in the field of cancer. MAML3 is a gene that encodes a protein known as MAML3, which is a member of the APR library gene family. This family of genes is known for its role in cell signaling and division, and MAML3 is thought to play a role in regulating cell proliferation and survival.

Recent studies have suggested that MAML3 may be a useful drug target or biomarker for a variety of diseases, including cancer. One reason for this is that MAML3 is known to be expressed in many different types of cancer, including breast, lung, and ovarian cancer. Additionally, studies have shown that MAML3 is involved in the development and progression of cancer, and that inhibiting its activity may be a effective way to treat cancer.

Another potential reason for targeting MAML3 is its role in cell signaling. MAML3 is a member of the APR library gene family, which is known for its role in cell signaling and division. This suggests that MAML3 may be involved in the regulation of cell signaling pathways, and that targeting its activity may be a way to interfere with these pathways. This could be useful for treating diseases that are caused by the disruption of normal cell signaling, such as cancer.

In addition to its potential as a drug target or biomarker, MAML3 is also of interest as a potential therapeutic agent for a variety of diseases. For example, studies have shown that MAML3 may be involved in the development and progression of certain types of cancer, and that inhibiting its activity may be a way to treat these diseases. Additionally, MAML3 has been shown to be involved in the regulation of cell growth and survival, which suggests that it may be a useful agent for treating diseases that are caused by the disruption of normal cell growth processes.

Overall, MAML3 is a gene that has the potential to be a drug target or biomarker for a variety of diseases. Its involvement in cell signaling and division, as well as its role in the development and progression of cancer, make it an attractive target for researchers to investigate further. Further studies are needed to determine the full extent of MAML3's potential as a drug target or biomarker, and to develop effective therapies for the treatment of cancer and other diseases.

Protein Name: Mastermind Like Transcriptional Coactivator 3

Functions: Acts as a transcriptional coactivator for NOTCH proteins. Has been shown to amplify NOTCH-induced transcription of HES1

The "MAML3 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about MAML3 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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