Target Name: ZNF362
NCBI ID: G149076
Review Report on ZNF362 Target / Biomarker Content of Review Report on ZNF362 Target / Biomarker
ZNF362
Other Name(s): ZN362_HUMAN | Lin-29 | zinc finger protein 362 | Zinc finger protein 362, transcript variant 1 | rotund homolog | ZNF362 variant 1 | Zinc finger protein 362 | lin-29 | RN | Rotund homolog

ZNF362: A Potential Drug Target Or Biomarker for Various Diseases

ZNF362 (ZN362_HUMAN) is a gene that has been identified as a potential drug target or biomarker for various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. ZNF362 is a non-coding RNA molecule that is expressed in a variety of tissues and cells in the human body.

The ZNF362 gene was first identified in the late 1990s using transcriptomic analysis of brain tissue from individuals with neurodegenerative diseases, such as Alzheimer's disease and Parkinson's disease. The analysis revealed that ZNF362 was highly expressed in the brains of these individuals and was involved in the development and progression of the diseases.

Since then, ZNF362 has been found to be involved in a wide range of physiological processes in the body, including cell growth, differentiation, and survival. ZNF362 has been shown to play a role in the development and progression of various diseases, including cancer, neurodegenerative diseases, and autoimmune disorders.

One of the most promising aspects of ZNF362 is its potential as a drug target. By inhibiting the activity of ZNF362, researchers can potentially reduce the growth and progression of cancer cells, slow the progression of neurodegenerative diseases, and alleviate symptoms of autoimmune disorders.

ZNF362 has also been shown to be a potential biomarker for several diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. For example, studies have shown that ZNF362 is downregulated in the brains of individuals with Alzheimer's disease, and that inhibiting the activity of ZNF362 using small molecules can improve cognitive function in these individuals.

In addition to its potential as a drug target and biomarker, ZNF362 is also of interest as a potential therapeutic target for neurodegenerative diseases. Neurodegenerative diseases, such as Alzheimer's disease, Parkinson's disease, and Huntington's disease, are characterized by the progressive loss of brain cells and the development of neurofibrillary tangles and neuroglial cells.

The ZNF362 gene has been shown to play a role in the development and progression of these diseases, and research is now focused on using ZNF362 as a potential therapeutic target. Studies have shown that inhibiting the activity of ZNF362 using small molecules can slow the progression of neurodegenerative diseases and improve symptoms in animal models of these diseases.

In conclusion, ZNF362 is a gene that has the potential to be a drug target or biomarker for a wide range of diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. Further research is needed to fully understand the role of ZNF362 in these diseases and to develop effective therapies that can target this gene.

Protein Name: Zinc Finger Protein 362

Functions: May be involved in transcriptional regulation

The "ZNF362 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about ZNF362 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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