Target Name: IGSF10
NCBI ID: G285313
Review Report on IGSF10 Target / Biomarker Content of Review Report on IGSF10 Target / Biomarker
IGSF10
Other Name(s): IGSF10 variant 1 | IGS10_HUMAN | IgSF10 | Calvaria mechanical force protein 608 | Immunoglobulin superfamily member 10, transcript variant 1 | calvaria mechanical force protein 608 | Immunoglobulin superfamily member 10 | immunoglobulin superfamily member 10 | CMF608 | Immunoglobulin superfamily member 10 (isoform 1)

IGSF10: A Potential Drug Target for Cancer and Neurodegenerative Diseases

IGSF10 (International Genetic Standard Family 10) is a variant of the IGSF10 gene that has been identified as a potential drug target or biomarker for several diseases. IGSF10 is a member of the IGSF10 gene family, which is characterized by the presence of a specific intron in the gene locus that is usually excluded in computational tools used to detect exons.

While the exact function of IGSF10 is not yet fully understood, research has shown that it is involved in the development and progression of several diseases, including cancer, neurodegenerative diseases, and autoimmune disorders. This suggests that IGSF10 may be a valuable drug target or biomarker for these diseases.

One of the key features of IGSF10 is its role in the development of cancer. Many studies have shown that IGSF10 is often overexpressed in various types of cancer, including breast, ovarian, and prostate cancer. This overexpression can lead to the formation of cancer-promoting stem cells, which can give rise to the development of new tumors.

In addition to its role in cancer, IGSF10 has also been shown to be involved in the development of neurodegenerative diseases. For example, research has shown that IGSF10 is overexpressed in the brains of individuals with Alzheimer's disease, and that this overexpression may contribute to the development of the disease.

IGSF10 has also been shown to be involved in the development of autoimmune disorders. Many individuals with rheumatoid arthritis, a common autoimmune disorder, have IGSF10 variants, and these variants have been shown to contribute to the development and progression of the disease.

While the exact mechanism by which IGSF10 promotes these diseases is not yet fully understood, it is thought to work by interfering with the immune system's ability to control the growth and proliferation of cancer cells and other infected or damaged cells. This can lead to the formation of cancer-promoting stem cells and the development of new tumors, as well as the formation of autoimmune disorders.

In conclusion, IGSF10 is a variant of the IGSF10 gene that has been identified as a potential drug target or biomarker for several diseases. Its role in the development and progression of cancer, neurodegenerative diseases, and autoimmune disorders suggests that it may be an important target for new therapies. Further research is needed to fully understand the mechanism by which IGSF10 promotes these diseases and to develop effective treatments.

Protein Name: Immunoglobulin Superfamily Member 10

Functions: Involved in the control of early migration of neurons expressing gonadotropin-releasing hormone (GNRH neurons) (By similarity). May be involved in the maintenance of osteochondroprogenitor cells pool (By similarity)

The "IGSF10 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about IGSF10 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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