Target Name: NCCRP1
NCBI ID: G342897
Review Report on NCCRP1 Target / Biomarker Content of Review Report on NCCRP1 Target / Biomarker
NCCRP1
Other Name(s): F-box only protein 50 | non-specific cytotoxic cell receptor protein 1 homolog | NCCRP1, F-box associated domain containing | FBXO50 | NCCRP-1 | nonspecific cytotoxic cell receptor protein 1 homolog | NCC receptor protein 1 homolog | FBX50_HUMAN

NCCRP1: A Potential Drug Target and Biomarker for the Treatment of Fibromyalgia

Fibromyalgia is a chronic pain condition characterized by widespread chronic pain, stiffness, and fatigue. It affects millions of people worldwide and can significantly impact their quality of life. The exact cause of fibromyalgia is not known, but it is thought to involve an overuse or overresponse of the immune system to various stimuli in the body.

NCCRP1: A Potential Drug Target and Biomarker

The immune system plays a crucial role in the development and progression of fibromyalgia. NCCRP1 is a non-coding RNA molecule that has been identified as a potential drug target and biomarker for fibromyalgia. NCCRP1 has been shown to regulate the immune response and is involved in the regulation of pain perception.

NCCRP1 gene expression and function

NCCRP1 is a gene that encodes a protein known as NCCRP1. The NCCRP1 protein is a 21-kDa protein that is expressed in various tissues and cells of the body. NCCRP1 has been shown to play a role in the regulation of immune cell function, cell death, and pain perception.

Studies have shown that NCCRP1 is involved in the regulation of immune cell function, including the regulation of T cell activity. NCCRP1 has also been shown to play a role in the regulation of cell death, including the regulation of apoptosis. Additionally, NCCRP1 has been shown to be involved in the regulation of pain perception, including the regulation of pain signaling.

Potential drug targeting

NCCRP1 has been identified as a potential drug target for the treatment of fibromyalgia due to its involvement in the regulation of immune cell function and pain perception. By targeting NCCRP1, researchers may be able to reduce the immune system's response to pain stimuli and improve pain relief in individuals with fibromyalgia.

NCCRP1 as a biomarker

NCCRP1 has also been identified as a potential biomarker for the diagnosis and monitoring of fibromyalgia. Fibromyalgia is often characterized by the presence of chronic pain, stiffness, and fatigue, which can be difficult to diagnose. NCCRP1 may be a useful biomarker for the diagnosis and monitoring of fibromyalgia as it is involved in the regulation of pain perception and immune cell function.

Conclusion

NCCRP1 is a non-coding RNA molecule that has been identified as a potential drug target and biomarker for the treatment of fibromyalgia. NCCRP1 is involved in the regulation of immune cell function, cell death, and pain perception, making it a promising target for the development of new treatments for fibromyalgia. Further research is needed to fully understand the role of NCCRP1 in fibromyalgia and to develop safe and effective treatments.

Protein Name: NCCRP1, F-box Associated Domain Containing

Functions: Promotes cell proliferation

The "NCCRP1 Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about NCCRP1 comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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