Target Name: GSTA7P
NCBI ID: G730152
Review Report on GSTA7P Target / Biomarker Content of Review Report on GSTA7P Target / Biomarker
GSTA7P
Other Name(s): GSTAP6 | GSTAP2 | glutathione S-transferase alpha 7, pseudogene | Glutathione S-transferase alpha 7, pseudogene

GSTA7P: A GPCR-Coupled Receptor Involved in Cell Signaling and Potential Drug Targets

GSTA7P (GSTAP6) is a protein that is expressed in various tissues throughout the body. It is a member of the G-protein-coupled receptor (GPCR) family, which is a large superfamily of transmembrane proteins that play a crucial role in cellular signaling . GSTA7P is known for its role in the regulation of cell signaling, particularly in the nervous system.

GSTA7P is a 7-transmembrane protein that is expressed in the brain, nervous system, and other tissues. It is characterized by a long extracellular domain that contains a conserved catalytic core and several unique features that are unique to this protein. The catalytic core of GSTA7P is a G-protein-coupled receptor that is responsible for transmitting signals from the cell surface to the intracellular signaling pathway.

One of the unique features of GSTA7P is its ability to interact with several different GPCRs, which allows it to play a critical role in the regulation of cell signaling. This is accomplished through its conserved catalytic core, which is capable of binding to a wide variety of ligands. In addition, GSTA7P has several unique features that are specific to this protein. These features include a N-terminal transmembrane domain that is rich in electrolytes, a unique carbohydrate ligand binding site, and a conserved C-terminal region that is involved in GST-伪 interactions.

GSTA7P is involved in a wide range of cellular signaling pathways, including neurotransmission, pain signaling, and cell survival. For example, GSTA7P is involved in the regulation of neurotransmitter release from axons, which is critical for the transmission of signals in the nervous system. It is also involved in the regulation of pain signaling, which is critical for the regulation of the body's immune response to pain. In addition, GSTA7P is involved in the regulation of cell survival, which is critical for the maintenance of normal cellular function.

GSTA7P is also a potential drug target (or biomarker) due to its involvement in several diseases, including cancer, neurodegenerative diseases, and autoimmune diseases. For example, GSTA7P has been shown to be involved in the regulation of cancer cell growth and metastasis. In In addition, GSTA7P has been shown to be involved in the regulation of neurodegenerative diseases, including Alzheimer's disease and Parkinson's disease.

In conclusion, GSTA7P is a unique and highly conserved protein that is involved in the regulation of cell signaling in the nervous system. Its ability to interact with several different GPCRs and its unique features make it a potential drug target (or biomarker) for the development of new treatments for a wide range of diseases. Further research is needed to fully understand the role of GSTA7P in cellular signaling and its potential as a drug target.

Protein Name: Glutathione S-transferase Alpha 7, Pseudogene

The "GSTA7P Target / Biomarker Review Report" is a customizable review of hundreds up to thousends of related scientific research literature by AI technology, covering specific information about GSTA7P comprehensively, including but not limited to:
•   general information;
•   protein structure and compound binding;
•   protein biological mechanisms;
•   its importance;
•   the target screening and validation;
•   expression level;
•   disease relevance;
•   drug resistance;
•   related combination drugs;
•   pharmacochemistry experiments;
•   related patent analysis;
•   advantages and risks of development, etc.
The report is helpful for project application, drug molecule design, research progress updates, publication of research papers, patent applications, etc. If you are interested to get a full version of this report, please feel free to contact us at BD@silexon.ai

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